Genetic Information

Gene & Transcript Details

Gene
MSH6
Transcript
NM_000179.3 MANE Select
Total Exons
Reference Sequence
NC_000002.11
Alternative Transcripts
IDStatusDetails
NM_000179.2 RefSeq Select 4435 nt | 153–4235
NM_000179.1 Alternative 4264 nt | 88–4170
NM_000179.3 MANE Select 4265 nt | 90–4172

Variant Details

HGVS Notation
NM_000179.3:c.-2G>T
Protein Change
?
Location
Exon 1 (Exon 1 of )
1
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.00145 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Uncertain Significance (VUS)
2 publications
Publications List
PMID: 26888055

The MSH6 c.-2G>T variant has been reported in the published literature in an individual with colorectal cancer (PMID: 34197922 (2021)) and in an individual with a family history of pancreatic cancer (PMID: 33939675 (2021)). The frequency of this variant in the general population (Genome Aggregation Database, http://gnomad.broadinstitute.org) is uninformative in the assessment of its pathogenicity. Based on the available information, we are unable to determine the clinical significance of this variant.

PMID: 26888055

Variant summary: MSH6 c.-2G>T is located in the untranslated mRNA region upstream of the initiation codon. The variant allele was found at a frequency of 1.2e-05 in 244064 control chromosomes. The available data on variant occurrences in the general population are insufficient to allow any conclusion about variant significance. c.-2G>T has been reported in the literature (Liu_2016). These report(s) do not provide unequivocal conclusions about association of the variant with Hereditary Nonpolyposis Colorectal Cancer. To our knowledge, no experimental evidence demonstrating an impact on protein function has been reported. The following publication have been ascertained in the context of this evaluation (PMID: 26888055). ClinVar contains an entry for this variant (Variation ID: 142219). Based on the evidence outlined above, the variant was classified as uncertain significance.

Clinical Statement

"This variant has been reported in ClinVar as Uncertain significance (8 clinical laboratories) and as Likely benign (5 clinical laboratories)."

COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene MSH6.

Functional Studies & Therapeutic Relevance

Functional Summary

The MSH6 -2G>T variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 135 bp
- Donor Loss (DL) 0.41 -1 bp
+ Acceptor Gain (AG) 0.0 -70 bp
+ Donor Gain (DG) 0.02 238 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines, the rule for PVS1 is: 'Very Strong Strength: Very Strong Nonsense/frameshift variant introducing Premature Termination Codon (PTC) a ≤ codon 1341 in MSH6.' The evidence for this variant shows: it is a noncoding 5′ UTR variant (c.-2G>T) not predicted to introduce a premature stop codon. Therefore, this criterion is not applied.

PS1

PS1 (Not Applied)

According to VCEP guidelines, the rule for PS1 is: 'Strong Strength: A predicted missense substitution that encodes the same amino acid change with a different underlying nucleotide change previously established by this VCEP as Pathogenic.' The evidence for this variant shows: it is not a missense variant and does not alter an amino acid. Therefore, this criterion is not applied.

PS2

PS2 (Not Applied)

According to VCEP guidelines, the rule for PS2 is: 'Very Strong Strength: Very Strong ≥ 4 de novo points.' The evidence for this variant shows: no de novo occurrence data are available. Therefore, this criterion is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines, the rule for PS3 is: 'Strong Strength: Strong Calibrated functional assays with functional odds for Pathogenicity > 18.7.' The evidence for this variant shows: no functional assay data exist for c.-2G>T. Therefore, this criterion is not applied.

PS4

PS4 (Not Applied)

According to standard ACMG guidelines, the rule for PS4 is: 'The prevalence of the variant in affected individuals is significantly increased compared with the prevalence in controls.' The evidence for this variant shows: no case-control or patient cohort data are available. Therefore, this criterion is not applied.

PM1

PM1 (Not Applied)

According to standard ACMG guidelines, the rule for PM1 is: 'Located in a mutational hot spot and/or critical and well-established functional domain without benign variation.' The evidence for this variant shows: c.-2G>T lies in the 5′ UTR, not in a known functional domain. Therefore, this criterion is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, the rule for PM2 is: 'Supporting Strength: Supporting Absent/extremely rare (<1 in 50,000 alleles) in gnomAD v4 dataset.' The evidence for this variant shows: observed at 4/275,398 alleles (MAF=0.00145%, ~1 in 68,850) which is below 1 in 50,000. Therefore, this criterion is applied at Supporting strength.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines, the rule for PM3 is: 'For recessive disorders, detected in trans with a pathogenic variant.' The evidence for this variant shows: no data on trans observations. Therefore, this criterion is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, the rule for PM4 is: 'Protein length changes due to in-frame deletions/insertions or stop-loss variants not in repetitive regions.' The evidence for this variant shows: c.-2G>T does not alter protein length. Therefore, this criterion is not applied.

PM5

PM5 (Not Applied)

According to standard ACMG guidelines, the rule for PM5 is: 'Novel missense change at an amino acid residue where a different missense change was classified as Pathogenic.' The evidence for this variant shows: it is not a missense variant. Therefore, this criterion is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, the rule for PM6 is: 'Assumed de novo, but without confirmation of paternity/maternity.' The evidence for this variant shows: no de novo data. Therefore, this criterion is not applied.

PP1

PP1 (Not Applied)

According to standard ACMG guidelines, the rule for PP1 is: 'Cosegregation with disease in multiple affected family members.' The evidence for this variant shows: no segregation data. Therefore, this criterion is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, the rule for PP2 is: 'Missense variant in a gene that has a low rate of benign missense variation and where missense variants are a common mechanism of disease.' The evidence for this variant shows: c.-2G>T is noncoding. Therefore, this criterion is not applied.

PP3

PP3 (Not Applied)

According to standard ACMG guidelines, the rule for PP3 is: 'Multiple lines of computational evidence support a deleterious effect.' The evidence for this variant shows: in silico prediction (CADD=1.04) is benign and SpliceAI score=0.41 is below high-confidence threshold for splicing; overall computational evidence is inconclusive. Therefore, this criterion is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines, the rule for PP4 is: 'Patient's phenotype or family history is highly specific for a disease with a single genetic etiology.' The evidence for this variant shows: no phenotype/tumor data. Therefore, this criterion is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, the rule for PP5 is: 'Reputable source recently reports variant as pathogenic but the evidence is not available to the laboratory to perform an independent evaluation.' The evidence for this variant shows: ClinVar submissions are VUS or Likely benign. Therefore, this criterion is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, the rule for BA1 is: 'Stand Alone Strength: GnomAD v4 Grpmax filtering allele frequency ≥ 0.0022.' The evidence for this variant shows: MAF=0.0000145 < 0.0022. Therefore, this criterion is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, the rule for BS1 is: 'Strong Strength: GnomAD v4 Grpmax filtering allele frequency ≥ 0.00022 and < 0.0022.' The evidence for this variant shows: MAF=0.0000145 < 0.00022. Therefore, this criterion is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, the rule for BS2 is: 'Strong Strength: Co-occurrence in trans with a known pathogenic variant in the same gene in a patient without CMMRD.' The evidence for this variant shows: no co-occurrence data. Therefore, this criterion is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, the rule for BS3 is: 'Strong Strength: Calibrated functional assays with functional odds for Pathogenicity ≤ 0.05.' The evidence for this variant shows: no functional assay data. Therefore, this criterion is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines, the rule for BS4 is: 'Strong Strength: Lack of co-segregation with disease in pedigree(s) with a combined Bayes LR h < 0.05.' The evidence for this variant shows: no segregation data. Therefore, this criterion is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, the rule for BP1 is: 'Missense variant in a gene for which primarily truncating variants are known to cause disease.' The evidence for this variant shows: it is noncoding. Therefore, this criterion is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, the rule for BP2 is: 'Observed in trans with a pathogenic variant in a patient with a recessive disorder or in cis with another pathogenic variant.' The evidence for this variant shows: no such observations. Therefore, this criterion is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, the rule for BP3 is: 'In-frame deletions/insertions in repetitive regions without known function.' The evidence for this variant shows: it is a single-nucleotide change in the UTR. Therefore, this criterion is not applied.

BP4

BP4 (Not Applied)

According to standard ACMG guidelines, the rule for BP4 is: 'Multiple lines of computational evidence suggest no impact on gene or gene product.' The evidence for this variant shows: conflicting in silico results (CADD benign, SpliceAI possible but below high-confidence threshold). Therefore, this criterion is not applied.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines, the rule for BP5 is: 'Variant found in a case with an alternate molecular basis for disease.' The evidence for this variant shows: no such case data. Therefore, this criterion is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, the rule for BP6 is: 'Reputable source recently reports variant as benign but the evidence is not available to the laboratory to perform an independent evaluation.' The evidence for this variant shows: ClinVar entries are conflicting (VUS/Likely benign) and not solely benign. Therefore, this criterion is not applied.

BP7

BP7 (Not Applied)

According to standard ACMG guidelines, the rule for BP7 is: 'A synonymous variant for which splicing prediction algorithms predict no impact on splicing.' The evidence for this variant shows: it is not synonymous but UTR. Therefore, this criterion is not applied.