Genetic Information

Gene & Transcript Details

Gene
TP53
Transcript
NM_000546.6 MANE Select
Total Exons
Reference Sequence
NC_000017.10
Alternative Transcripts
IDStatusDetails
NM_000546.5 RefSeq Select 2591 nt | 203–1384
NM_000546.3 Alternative 2640 nt | 252–1433
NM_000546.6 MANE Select 2512 nt | 143–1324
NM_000546.4 Alternative 2586 nt | 198–1379
NM_000546.2 Alternative 2629 nt | 252–1433

Variant Details

HGVS Notation
NM_000546.6:c.215_216delinsG
Protein Change
P72Rfs*51
Location
Exon 4 (Exon 4 of )
4
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene TP53.

Functional Studies & Therapeutic Relevance

Functional Summary

The TP53 P72Rfs*51 variant is a truncating mutation in the TP53 gene, a critical tumor suppressor involved in the DNA damage response pathway. Functional evidence indicates that truncating mutations in TP53 lead to the production of C-terminally truncated proteins, which are predicted to be inactivating. These alterations have been experimentally shown to promote cancer cell proliferation, survival, and metastasis, partly due to aberrant mitochondrial localization and regulation of survival-related genes. Therefore, the TP53 P72Rfs*51 variant is functionally characterized as likely damaging.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Unknown
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 100 bp
- Donor Loss (DL) 0.05 41 bp
+ Acceptor Gain (AG) 0.01 -85 bp
+ Donor Gain (DG) 0.01 -159 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Very Strong)

According to VCEP guidelines for PVS1: "Nonsense or frameshift variants predicted to result in nonsense-mediated decay (NMD) for nonsense variants upstream of p.Lys351 and for frameshift induced premature termination codon (PTC) upstream of p.Lys351: PVS1 (Very Strong Strength)". The evidence for this variant shows: the c.215_216delCCinsG (P72Rfs*51) creates a frameshift resulting in a premature stop codon upstream of p.Lys351, predicted to trigger NMD. Therefore, this criterion is applied at Very Strong strength because it meets the VCEP conditions for NMD upstream of p.Lys351.

PS1

PS1 (Not Applied)

According to VCEP guidelines for PS1: "Can be applied to variants asserted as Pathogenic or Likely Pathogenic following TP53 VCEP’s specifications" for missense variants. The evidence for this variant shows: c.215_216delCCinsG is a frameshift, not a missense. Therefore, this criterion is not applied.

PS2

PS2 (Not Applied)

According to VCEP guidelines for PS2: point system based on confirmed de novo events. The evidence for this variant shows: no de novo data available. Therefore, this criterion is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines caveat: "PS3 should not be applied at any strength if PVS1 is applied at full strength." The evidence for this variant shows: PVS1_Very Strong has been applied. Therefore, PS3 is not applied.

PS4

PS4 (Not Applied)

According to standard ACMG guidelines for PS4: "Increased prevalence in affected individuals over controls" with a point system. The evidence for this variant shows: no case-control or proband point data. Therefore, this criterion is not applied.

PM1

PM1 (Not Applied)

According to VCEP guidelines for PM1: "Missense variants within hotspot codons". The evidence for this variant shows: it is a frameshift variant, not missense. Therefore, this criterion is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines for PM2: "This rule should be applied at supporting level. Variant should have an allele frequency of less than 0.00003 (0.003%) in gnomAD”. The evidence for this variant shows: absent from gnomAD (MAF=0%). Therefore, this criterion is applied at Supporting strength because the variant’s frequency is below the threshold.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines for PM3: "For variants in trans with a pathogenic variant in recessive disorders". The evidence for this variant shows: not evaluated in a recessive context. Therefore, this criterion is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines for PM4: "Protein length changes due to in-frame indels or stop-loss variants". The evidence for this variant shows: it is a frameshift predicted to cause NMD, covered by PVS1. Therefore, this criterion is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines for PM5: "Missense variant at an amino acid residue where pathogenic missense changes were seen". The evidence for this variant shows: it is a frameshift, not missense. Therefore, this criterion is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines for PM6: "Assumed de novo without confirmation of paternity/maternity". The evidence for this variant shows: no de novo information. Therefore, this criterion is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines for PP1: "Cosegregation in families". The evidence for this variant shows: no segregation data available. Therefore, this criterion is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines for PP2: "Missense variant in a gene with low rate of benign missense variants". The evidence for this variant shows: it is a frameshift. Therefore, this criterion is not applied.

PP3

PP3 (Not Applied)

According to VCEP guidelines caveat: "PP3 should not be used in combination with PVS1." The evidence for this variant shows: PVS1 applied. Therefore, this criterion is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines for PP4: "Phenotype specificity". The evidence for this variant shows: no phenotype specificity data. Therefore, this criterion is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines for PP5: "Reputable source reports variant as pathogenic". The evidence for this variant shows: no such reports. Therefore, this criterion is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines for BA1: "Stand-alone benign filtering allele frequency ≥0.001". The evidence for this variant shows: absent in population. Therefore, this criterion is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines for BS1: "Filtering allele frequency ≥0.0003 but <0.001". The evidence for this variant shows: absent in population. Therefore, this criterion is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines for BS2: "Observation in ≥8 unaffected older females". The evidence for this variant shows: no such data. Therefore, this criterion is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines for BS3: "Functional data shows no loss of function". The evidence for this variant shows: truncating predicted LOF and PVS1 applied. Therefore, this criterion is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines for BS4: "Lack of segregation in affected members". The evidence for this variant shows: no segregation data. Therefore, this criterion is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines for BP1: "Missense variant in gene where only LOF causes disease". The evidence for this variant shows: it is a LOF variant. Therefore, this criterion is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines for BP2: "Observed in trans with a pathogenic variant for a dominant disorder". The evidence for this variant shows: not applicable. Therefore, this criterion is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines for BP3: "In-frame indels in repetitive region without functional impact". The evidence for this variant shows: frameshift variant. Therefore, this criterion is not applied.

BP4

BP4 (Not Applied)

According to VCEP guidelines for BP4: "Computational evidence supports benign effect". The evidence for this variant shows: truncating variant, PVS1 applied. Therefore, this criterion is not applied.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines for BP5: "Variant found in case with an alternate cause". The evidence for this variant shows: no data. Therefore, this criterion is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines for BP6: "Reputable source reports variant as benign". The evidence for this variant shows: no such reports. Therefore, this criterion is not applied.

BP7

BP7 (Not Applied)

According to VCEP guidelines for BP7: "Synonymous or intronic variant with no splicing impact". The evidence for this variant shows: not applicable (frameshift). Therefore, this criterion is not applied.