Genetic Information

Gene & Transcript Details

Gene
KRAS
Transcript
NM_033360.4 MANE Select
Total Exons
Reference Sequence
NC_000012.11
Alternative Transcripts
IDStatusDetails
NM_033360.2 Alternative 5436 nt | 182–751
NM_033360.4 Alternative 5430 nt | 191–760
NM_033360.3 Alternative 5889 nt | 193–762

Variant Details

HGVS Notation
NM_033360.4:c.93A>G
Protein Change
E31=
Location
Exon 2 (Exon 2 of )
2
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0018 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Uncertain Significance (VUS)
2 publications
Clinical Statement

"This variant has been reported in ClinVar as Likely benign (5 clinical laboratories) and as Uncertain significance (1 clinical laboratories)."

COSMIC Somatic Evidence

Open
COSMIC ID
COSM9312101
Recurrence
1 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene KRAS.

Functional Studies & Therapeutic Relevance

Functional Summary

The variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 -441 bp
- Donor Loss (DL) 0.0 206 bp
+ Acceptor Gain (AG) 0.0 -469 bp
+ Donor Gain (DG) 0.0 1 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to standard ACMG guidelines the rule for PVS1 is: "Null variant (nonsense, frameshift, start codon, splicing +1/2, startgain, single or multi-exon deletion) in a gene where LOF is a known mechanism of disease." The evidence for this variant shows: it is a synonymous (silent) variant, not predicted to produce a null allele. Therefore, this criterion is not applied.

PS1

PS1 (Not Applied)

According to VCEP guidelines the rule for PS1 is: "Strong Same amino acid change as a previously established pathogenic variant regardless of nucleotide change (including analogous residues in HRAS, KRAS, MRAS, NRAS, RIT1, RRAS2)." The evidence for this variant shows: it is a synonymous change (E31=), so no amino acid change. Therefore, this criterion is not applied.

PS2

PS2 (Not Applied)

According to VCEP guidelines the rule for PS2 is: "Very Strong if de novo (both maternity and paternity confirmed) or Strong/Moderate if de novo with varying points." The evidence for this variant shows: de novo status is unknown. Therefore, this criterion is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines the rule for PS3 is: "Moderate if two or more different approved assays show a deleterious effect; Supporting if one approved assay." The evidence for this variant shows: no functional studies have been performed. Therefore, this criterion is not applied.

PS4

PS4 (Not Applied)

According to VCEP guidelines the rule for PS4 is: "Strong if ≥5 proband points; Moderate if ≥3; Supporting if ≥1." The evidence for this variant shows: no case‐control or proband data supporting enrichment in affected individuals. Therefore, this criterion is not applied.

PM1

PM1 (Not Applied)

According to VCEP guidelines the rule for PM1 is: "Moderate for variants in critical functional domains (P-loop AA10-17, SW1 AA25-40, SW2 AA57-64, SAK AA145-156)." The evidence for this variant shows: it is synonymous and does not alter amino acid sequence within SW1. Therefore, this criterion is not applied.

PM2

PM2 (Not Applied)

According to VCEP guidelines the rule for PM2 is: "Supporting: The variant must be absent from controls (gnomAD)." The evidence for this variant shows: it is present in gnomAD at MAF 0.0018%. Therefore, this criterion is not applied.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines the rule for PM3 is: "For recessive disorders, detected in trans with a pathogenic variant." The evidence for this variant shows: no evidence of trans configuration in a recessive context. Therefore, this criterion is not applied.

PM4

PM4 (Not Applied)

According to VCEP guidelines the rule for PM4 is: "Moderate for protein length changes due to in-frame deletions/insertions outside repetitive regions." The evidence for this variant shows: it is synonymous with no indel. Therefore, this criterion is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines the rule for PM5 is: "Strong if ≥2 different known pathogenic residue changes at the same codon in ≥5 probands; Moderate if 1 pathogenic residue change." The evidence for this variant shows: it is synonymous and does not change the residue. Therefore, this criterion is not applied.

PM6

PM6 (Not Applied)

According to VCEP guidelines the rule for PM6 is: "Strong/Moderate/Supporting for unconfirmed de novo occurrences with varying point values." The evidence for this variant shows: de novo status is unknown. Therefore, this criterion is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines the rule for PP1 is: "Supporting/Moderate/Strong based on number of informative meioses." The evidence for this variant shows: no segregation data available. Therefore, this criterion is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines the rule for PP2 is: "Supporting for missense variants in a gene with low rate of benign missense variants and where missense is a common mechanism." The evidence for this variant shows: it is synonymous, not missense. Therefore, this criterion is not applied.

PP3

PP3 (Not Applied)

According to VCEP guidelines the rule for PP3 is: "Supporting for missense variants with REVEL ≥0.7 or splicing predictions matching disease mechanism." The evidence for this variant shows: it is synonymous and splicing predictors show no impact. Therefore, this criterion is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines the rule for PP4 is: "Supporting when patient phenotype is highly specific for a single gene disorder." The evidence for this variant shows: no patient phenotype data provided. Therefore, this criterion is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines the rule for PP5 is: "Supporting when a reputable source reports the variant as pathogenic without primary evidence." The evidence for this variant shows: ClinVar entries are conflicting (Likely benign and VUS). Therefore, this criterion is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines the rule for BA1 is: "Stand Alone if gnomAD filtering allele frequency ≥0.05%." The evidence for this variant shows: MAF = 0.0018%, below threshold. Therefore, this criterion is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines the rule for BS1 is: "Strong if gnomAD filtering allele frequency ≥0.025%." The evidence for this variant shows: MAF = 0.0018%, below threshold. Therefore, this criterion is not applied.

BS2

BS2 (Not Applied)

According to standard ACMG guidelines the rule for BS2 is: "Strong/Supporting when observed in healthy adult individuals for disorders with full penetrance." The evidence for this variant shows: no observations in well‐characterized healthy cohorts. Therefore, this criterion is not applied.

BS3

BS3 (Not Applied)

According to standard ACMG guidelines the rule for BS3 is: "Strong when well‐established functional studies show no damaging effect." The evidence for this variant shows: no functional study data. Therefore, this criterion is not applied.

BS4

BS4 (Not Applied)

According to standard ACMG guidelines the rule for BS4 is: "Strong when lack of segregation in affected members of a family." The evidence for this variant shows: segregation data unavailable. Therefore, this criterion is not applied.

BP1

BP1 (Not Applied)

According to VCEP guidelines the rule for BP1 is: "Supporting for truncating variants in genes where gain-of-function is disease mechanism." The evidence for this variant shows: it is synonymous, not truncating. Therefore, this criterion is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines the rule for BP2 is: "Supports benign when observed in trans with a pathogenic variant for a dominant disorder or cis with another pathogenic variant for recessive." The evidence for this variant shows: no such observations. Therefore, this criterion is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines the rule for BP3 is: "Supporting when in-frame indels in repetitive regions without known function." The evidence for this variant shows: it is synonymous, not an indel. Therefore, this criterion is not applied.

BP4

BP4 (Not Applied)

According to VCEP guidelines the rule for BP4 is: "Supporting for missense variants with REVEL ≤0.3." The evidence for this variant shows: it is synonymous, not missense, despite benign computational predictions. Therefore, this criterion is not applied.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines the rule for BP5 is: "Supporting when a variant is found in a case with an alternate molecular basis for disease." The evidence for this variant shows: no such cases reported. Therefore, this criterion is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines the rule for BP6 is: "Supporting when a reputable source reports the variant as benign without evidence." The evidence for this variant shows: ClinVar submissions include Likely benign but with conflicting calls. Therefore, this criterion is not applied.

BP7

BP7 (Supporting)

According to VCEP guidelines the rule for BP7 is: "Supporting A synonymous (silent) variant for which splicing prediction algorithms predict no impact to the splice consensus sequence nor the creation of a new splice site AND the nucleotide is not highly conserved." The evidence for this variant shows: it is synonymous (E31=), SpliceAI predicts no impact on splicing, and the nucleotide is not highly conserved. Therefore, this criterion is applied at Supporting strength.