Genetic Information

Gene & Transcript Details

Gene
PIK3CA
Transcript
NM_006218.4 MANE Select
Total Exons
Reference Sequence
NC_000003.11
Alternative Transcripts
IDStatusDetails
NM_006218.2 Alternative 3724 nt | 158–3364
NM_006218.3 Alternative 9104 nt | 158–3364
NM_006218.4 MANE Select 9259 nt | 324–3530

Variant Details

HGVS Notation
NM_006218.4:c.3006C>A
Protein Change
F1002L
Location
Exon 21 (Exon 21 of )
21
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
COSM8824511
Recurrence
2 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
Accessing full COSMIC database details requires institutional login or subscription.

Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene PIK3CA.

Functional Studies & Therapeutic Relevance

Functional Summary

The PIK3CA F1002L variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.445
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 -69 bp
- Donor Loss (DL) 0.0 -121 bp
+ Acceptor Gain (AG) 0.0 -62 bp
+ Donor Gain (DG) 0.0 459 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to standard ACMG guidelines, the rule for PVS1 is: null variant (nonsense, frameshift, canonical ±1 or 2 splice sites, initiation codon, single exon or multiexon deletion) in a gene where loss of function is a known mechanism of disease. The evidence for this variant shows: a missense change (F1002L) not predicted to cause loss of function. Therefore, this criterion is not applied.

PS1

PS1 (Not Applied)

According to standard ACMG guidelines, the rule for PS1 is: same amino acid change as a previously established pathogenic variant regardless of nucleotide change. The evidence for this variant shows: no previously established pathogenic variant resulting in F1002L. Therefore, this criterion is not applied.

PS2

PS2 (Not Applied)

According to VCEP guidelines, PS2 requires de novo occurrence with confirmed maternity/paternity and/or tissue-specific evidence for somatic contexts. The evidence for this variant shows: no data on parental or tissue segregation. Therefore, this criterion is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines, PS3 requires well-validated functional assays demonstrating a deleterious effect. The evidence for this variant shows: no functional studies available for F1002L. Therefore, this criterion is not applied.

PS4

PS4 (Not Applied)

According to VCEP guidelines, PS4 requires quantitative case/phenotype data (absent from controls per PM2) with point-based scoring. The evidence for this variant shows: no reported cases or phenotype data. Therefore, this criterion is not applied.

PM1

PM1 (Not Applied)

According to VCEP guidelines, PM1 Supporting is for residues affecting critical functional domains provided in Table 4. The evidence for this variant shows: F1002 is not within a VCEP-defined hotspot or critical domain. Therefore, this criterion is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, the rule for PM2 is: Supporting Absent/rare from controls in an ethnically-matched cohort population sample (≥1). The evidence for this variant shows: absent from gnomAD and other population databases (MAF=0%). Therefore, this criterion is applied at Supporting strength because the variant is absent from controls.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines, the rule for PM3 is: detected in trans with a pathogenic variant for a recessive disorder. The evidence for this variant shows: insufficient data on trans observations and PIK3CA is not a recessive gene. Therefore, this criterion is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, the rule for PM4 is: protein length changes due to in-frame indels or stop-loss variants. The evidence for this variant shows: a single amino acid substitution without length change. Therefore, this criterion is not applied.

PM5

PM5 (Not Applied)

According to standard ACMG guidelines, the rule for PM5 is: novel missense change at an amino acid residue where a different missense change is pathogenic. The evidence for this variant shows: no other pathogenic missense at F1002. Therefore, this criterion is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, the rule for PM6 is: assumed de novo without confirmation of paternity/maternity. The evidence for this variant shows: no de novo data. Therefore, this criterion is not applied.

PP1

PP1 (Not Applied)

According to standard ACMG guidelines, the rule for PP1 is: cosegregation with disease in multiple affected family members. The evidence for this variant shows: no segregation data. Therefore, this criterion is not applied.

PP2

PP2 (Supporting)

According to VCEP guidelines, the rule for PP2 is: Supporting Missense constraint computed in ExAC/gnomAD was utilized; award if z-score > 3.09 for PIK3CA. The evidence for this variant shows: PIK3CA has a missense z-score > 3.09 and the variant is a missense. Therefore, this criterion is applied at Supporting strength because PIK3CA is constrained and the variant is missense.

PP3

PP3 (Not Applied)

According to standard ACMG guidelines, the rule for PP3 is: multiple lines of computational evidence support a deleterious effect. The evidence for this variant shows: conflicting in silico predictions with a REVEL of 0.45 and SpliceAI predicting no impact. Therefore, this criterion is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines, the rule for PP4 is: patient’s phenotype or family history highly specific for a disease with a single genetic etiology. The evidence for this variant shows: no phenotype data. Therefore, this criterion is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, the rule for PP5 is: reputable source classifies variant as pathogenic. The evidence for this variant shows: no entries in ClinVar or other expert databases. Therefore, this criterion is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, the rule for BA1 is: Stand Alone allele frequency > 0.0926%. The evidence for this variant shows: MAF=0% in population databases. Therefore, this criterion is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, the rule for BS1 is: Strong allele frequency > 0.0185%. The evidence for this variant shows: MAF=0%. Therefore, this criterion is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, the rule for BS2 is: Strong observation in ≥3 healthy adult individuals. The evidence for this variant shows: no homozygotes or multiple healthy carriers documented. Therefore, this criterion is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, the rule for BS3 is: Strong well-validated functional studies showing no damaging effect. The evidence for this variant shows: no functional assays. Therefore, this criterion is not applied.

BS4

BS4 (Not Applied)

According to standard ACMG guidelines, the rule for BS4 is: lack of segregation in affected members. The evidence for this variant shows: no segregation data. Therefore, this criterion is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, the rule for BP1 is: missense in a gene where only truncating variants cause disease. The evidence for this variant shows: PIK3CA disease is driven by activating missense variants. Therefore, this criterion is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, the rule for BP2 is: observed in cis or trans with a pathogenic variant. The evidence for this variant shows: no data on cis/trans occurrence. Therefore, this criterion is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, the rule for BP3 is: in-frame indel in a repetitive region without known function. The evidence for this variant shows: it is a missense, not an indel. Therefore, this criterion is not applied.

BP4

BP4 (Not Applied)

According to VCEP guidelines, BP4 applies only to synonymous, intronic, or UTR variants with splicing predictions. The evidence for this variant shows: a missense change with conflicting computational predictions. Therefore, this criterion is not applied.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines, the rule for BP5 is: variant found in a case with an alternate molecular basis for disease. The evidence for this variant shows: no evidence of another molecular diagnosis. Therefore, this criterion is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, the rule for BP6 is: reputable source classifies variant as benign. The evidence for this variant shows: no such classifications in ClinVar or other databases. Therefore, this criterion is not applied.

BP7

BP7 (Not Applied)

According to standard ACMG guidelines, the rule for BP7 is: synonymous or intronic variant with no splicing impact and low conservation. The evidence for this variant shows: it is missense. Therefore, this criterion is not applied.