Genetic Information

Gene & Transcript Details

Gene
PALB2
Transcript
NM_024675.4 MANE Select
Total Exons
Reference Sequence
NC_000016.9
Alternative Transcripts
IDStatusDetails
NM_024675.4 MANE Select 4008 nt | 154–3714
NM_024675.3 RefSeq Select 4069 nt | 201–3761

Variant Details

HGVS Notation
NM_024675.4:c.3351-16_3351-14del
Protein Change
?
Location
Exon 12 (Exon 12 of )
12
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
Accessing full COSMIC database details requires institutional login or subscription.

Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene PALB2.

Functional Studies & Therapeutic Relevance

Functional Summary

The variant PALB2 3351-16_3351-14del has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.01 -13 bp
- Donor Loss (DL) 0.02 118 bp
+ Acceptor Gain (AG) 0.0 366 bp
+ Donor Gain (DG) 0.0 -95 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines, the PVS1 rule applies to null variants in PALB2 per the PVS1 decision tree. The evidence for this variant shows it is a small intronic deletion outside canonical splice sites, not predicted to cause loss of function. Therefore, this criterion is not applied at any strength because the variant does not meet PVS1 requirements.

PS1

PS1 (Not Applied)

According to VCEP guidelines, PS1 applies when a variant results in the same amino acid change as a known pathogenic variant. The evidence for this variant shows no change in protein sequence is predicted. Therefore, PS1 is not applied.

PS2

PS2 (Not Applied)

According to standard ACMG guidelines, PS2 applies to confirmed de novo variants. No de novo data are available for this variant. Therefore, PS2 is not applied.

PS3

PS3 (Not Applied)

According to standard ACMG guidelines, PS3 requires well-established functional studies showing a damaging effect. No functional studies have been performed for this variant. Therefore, PS3 is not applied.

PS4

PS4 (Not Applied)

According to VCEP guidelines, PS4 applies to case-control data meeting specific statistical thresholds. No case-control or cohort data are available for this variant. Therefore, PS4 is not applied.

PM1

PM1 (Not Applied)

According to standard ACMG guidelines, PM1 applies to variants located in mutational hotspots or critical functional domains without benign variation. This variant is intronic outside known functional domains. Therefore, PM1 is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, PM2: Supporting Variant absent in gnomAD or present in ≤ 1/300,000 alleles. The evidence for this variant shows it is not found in gnomAD (MAF=0%). Therefore, PM2 is applied at Supporting strength because the variant is absent from population databases.

PM3

PM3 (Not Applied)

According to VCEP guidelines, PM3 applies to recessive inheritance contexts with trans observations. No recessive or in trans data are available for this variant. Therefore, PM3 is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, PM4 applies to protein length changes due to in-frame indels or stop-loss. This variant is intronic and does not affect protein length. Therefore, PM4 is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines, PM5 applies to frameshifting or truncating variants with premature stop codons upstream of known pathogenic variants. This variant is intronic and does not create a termination codon. Therefore, PM5 is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, PM6 applies to assumed de novo variants without parental confirmation. There is no data suggesting de novo occurrence. Therefore, PM6 is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines, PP1 applies for segregation data in families. No segregation data are available. Therefore, PP1 is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, PP2 applies to missense variants in genes where benign missense variants are rare. This variant is intronic and not a missense change. Therefore, PP2 is not applied.

PP3

PP3 (Not Applied)

According to VCEP guidelines, PP3 applies when in silico tools predict impact on splicing or protein. SpliceAI predicts no impact (max score 0.02) and other predictors are benign. Therefore, PP3 is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines, PP4 applies to highly specific phenotypes. No patient phenotype data are provided. Therefore, PP4 is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, PP5 applies to reputable source assertions of pathogenicity. This variant is not in ClinVar or similar databases. Therefore, PP5 is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, BA1 applies for allele frequency >0.1% in gnomAD. This variant is absent from population databases. Therefore, BA1 is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, BS1 applies for allele frequency greater than expected for disease (>0.01%). This variant is absent in gnomAD. Therefore, BS1 is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, BS2 applies for observations in healthy adults per Fanconi Anemia BS2 tables. No such data exist. Therefore, BS2 is not applied.

BS3

BS3 (Not Applied)

According to standard ACMG guidelines, BS3 requires well-established functional studies showing no damaging effect. No functional data are available. Therefore, BS3 is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines, BS4 applies for lack of segregation (LOD≤–1.28). No segregation data are available. Therefore, BS4 is not applied.

BP1

BP1 (Not Applied)

According to VCEP guidelines, BP1 applies to all missense variants in certain contexts. This variant is intronic. Therefore, BP1 is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, BP2 applies when observations in trans with a pathogenic variant for a dominant disorder. No such data exist. Therefore, BP2 is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, BP3 applies to in-frame indels in repetitive regions. This variant is intronic. Therefore, BP3 is not applied.

BP4

BP4 (Supporting)

According to VCEP guidelines, BP4: RNA: at least one well-established in silico predictor shows no impact on splicing. SpliceAI predicts no splicing impact (max score 0.02) and other predictors are benign. Therefore, BP4 is applied at Supporting strength because computational evidence supports no impact.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines, BP5 applies when variant found in a gene for which alternate molecular basis is established. Not applicable here. Therefore, BP5 is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, BP6 applies to reputable source assertions of benign impact. None exist. Therefore, BP6 is not applied.

BP7

BP7 (Not Applied)

According to VCEP guidelines, BP7 applies to synonymous or deep intronic variants with observed lack of RNA defect. No RNA assay data are available. Therefore, BP7 is not applied.