Genetic Information

Gene & Transcript Details

Gene
MSH6
Transcript
NM_000179.3 MANE Select
Total Exons
Reference Sequence
NC_000002.11
Alternative Transcripts
IDStatusDetails
NM_000179.3 MANE Select 4265 nt | 90–4172
NM_000179.2 RefSeq Select 4435 nt | 153–4235
NM_000179.1 Alternative 4264 nt | 88–4170

Variant Details

HGVS Notation
NM_000179.3:c.1564_1565del
Protein Change
Q522Dfs*8
Location
Exon 4 (Exon 4 of )
4
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene MSH6.

Functional Studies & Therapeutic Relevance

Functional Summary

The MSH6 Q522Dfs*8 variant is a truncating mutation that results in a premature stop codon, leading to a C-terminally truncated protein. This truncation causes the loss of part or all of the highly conserved MutS domain, resulting in impaired ATPase activity, DNA binding, and mismatch repair. Such defects are associated with microsatellite instability and hypermutation, particularly in colorectal and endometrial cancers. Therefore, functional evidence supports that this variant likely has a damaging effect on the protein's function.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Unknown
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.02 494 bp
- Donor Loss (DL) 0.05 46 bp
+ Acceptor Gain (AG) 0.0 -448 bp
+ Donor Gain (DG) 0.04 -276 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Very Strong)

According to VCEP guidelines, the rule for PVS1 is: "Very Strong Nonsense/frameshift variant introducing Premature Termination Codon (PTC) ≤ codon 1341 in MSH6." The evidence for this variant shows: c.1564_1565delCA (p.Q522Dfs*8) introduces a premature stop at codon 529, well before codon 1341. Therefore, this criterion is applied at Very Strong strength because the variant is a frameshift leading to a premature termination codon in a gene where loss-of-function is a known mechanism of disease.

PS1

PS1 (Not Applied)

According to VCEP guidelines, the rule for PS1 is: "A predicted missense substitution encoding the same amino acid change as a previously established pathogenic variant." The evidence for this variant shows: it is a frameshift, not a missense substitution. Therefore, this criterion is not applied.

PS2

PS2 (Not Applied)

According to standard ACMG guidelines, the rule for PS2 is: "Confirmed de novo (both maternity and paternity confirmed) in a patient with the disease and no family history." The evidence for this variant shows: no data on de novo occurrence. Therefore, this criterion is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines, the rule for PS3 Strong is: "Calibrated functional assays with functional odds for pathogenicity >18.7." The evidence for this variant shows: published studies describe loss of mismatch repair activity but provide no calibrated functional odds. Therefore, this criterion is not applied because the functional assay data are not calibrated to meet the VCEP threshold.

PS4

PS4 (Not Applied)

According to standard ACMG guidelines, the rule for PS4 is: "The prevalence of the variant in affected individuals is significantly increased compared to controls." The evidence for this variant shows: no case-control or statistical association data. Therefore, this criterion is not applied.

PM1

PM1 (Not Applied)

According to standard ACMG guidelines, the rule for PM1 is: "Located in a mutational hot spot and/or critical and well-established functional domain without benign variation." The evidence for this variant shows: insufficient data on specific domain boundaries or hotspot status beyond LOF. Therefore, this criterion is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, the rule for PM2 is: "Absent/extremely rare (<1 in 50,000 alleles) in gnomAD v4 dataset." The evidence for this variant shows: MAF=0% in gnomAD. Therefore, this criterion is applied at Supporting strength because the variant is absent from large population databases.

PM3

PM3 (Not Applied)

According to VCEP guidelines, the rule for PM3 is: "Observed in trans with a pathogenic variant in a recessive disease context." The evidence for this variant shows: no data on trans observations with another pathogenic MSH6 variant. Therefore, this criterion is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, the rule for PM4 is: "Protein length changes due to in-frame deletions/insertions in a non-repeat region or stop-loss variants." The evidence for this variant shows: it is a frameshift leading to truncation, covered by PVS1. Therefore, this criterion is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines, the rule for PM5 is: "Missense change at an amino acid residue where a different missense change was classified as pathogenic." The evidence for this variant shows: it is a frameshift, not a missense change. Therefore, this criterion is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, the rule for PM6 is: "Assumed de novo, without confirmation of paternity and maternity." The evidence for this variant shows: no de novo data available. Therefore, this criterion is not applied.

PP1

PP1 (Not Applied)

According to standard ACMG guidelines, the rule for PP1 is: "Co-segregation with disease in multiple affected family members." The evidence for this variant shows: no segregation data provided. Therefore, this criterion is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, the rule for PP2 is: "Missense variant in a gene with a low rate of benign missense variation and where missense variants are a common mechanism of disease." The evidence for this variant shows: it is a frameshift. Therefore, this criterion is not applied.

PP3

PP3 (Not Applied)

According to standard ACMG guidelines, the rule for PP3 is: "Multiple lines of computational evidence support a deleterious effect on the gene or gene product." The evidence for this variant shows: in silico predictions were minimal and the variant is a frameshift. Therefore, this criterion is not applied.

PP4

PP4 (Not Applied)

According to VCEP guidelines, the rule for PP4 is: "Tumor characteristics (e.g., MSI-H, loss of MMR protein) consistent with MSH6 variant pathogenicity." The evidence for this variant shows: no tumor phenotype or MSI data provided. Therefore, this criterion is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, the rule for PP5 is: "Reputable source reports variant as pathogenic without available evidence." The evidence for this variant shows: not reported in ClinVar or similar. Therefore, this criterion is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, the rule for BA1 is: "GnomAD v4 Grpmax filtering allele frequency ≥0.0022." The evidence for this variant shows: MAF=0%. Therefore, this criterion is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, the rule for BS1 is: "GnomAD v4 Grpmax filtering allele frequency ≥0.00022 and <0.0022." The evidence for this variant shows: MAF=0%. Therefore, this criterion is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, the rule for BS2 is: "Co-occurrence in trans with a known pathogenic variant in LS cancer without CMMRD phenotype." The evidence for this variant shows: no co-occurrence data. Therefore, this criterion is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, the rule for BS3 is: "Calibrated functional assays with functional odds for pathogenicity ≤0.05 or synonymous/intronic variants with no splicing impact." The evidence for this variant shows: no calibrated benign functional assays. Therefore, this criterion is not applied.

BS4

BS4 (Not Applied)

According to standard ACMG guidelines, the rule for BS4 is: "Lack of segregation in affected family members." The evidence for this variant shows: no segregation data. Therefore, this criterion is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, the rule for BP1 is: "Missense variant in a gene where only truncating variants cause disease." The evidence for this variant shows: it is a truncating variant. Therefore, this criterion is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, the rule for BP2 is: "Observed in cis with a pathogenic variant or in trans in a dominant disorder." The evidence for this variant shows: no such observations. Therefore, this criterion is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, the rule for BP3 is: "In-frame deletions/insertions in a repetitive region without a known function." The evidence for this variant shows: it is a frameshift, not an in-frame event. Therefore, this criterion is not applied.

BP4

BP4 (Not Applied)

According to standard ACMG guidelines, the rule for BP4 is: "Multiple lines of computational evidence suggest no impact on gene or gene product." The evidence for this variant shows: frameshift leading to LOF. Therefore, this criterion is not applied.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines, the rule for BP5 is: "Variant found in a case with an alternate molecular basis for disease." The evidence for this variant shows: no alternate molecular basis reported. Therefore, this criterion is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, the rule for BP6 is: "Reputable source reports variant as benign without evidence." The evidence for this variant shows: no such report. Therefore, this criterion is not applied.

BP7

BP7 (Not Applied)

According to standard ACMG guidelines, the rule for BP7 is: "Synonymous variant with no splicing impact." The evidence for this variant shows: it is a frameshift. Therefore, this criterion is not applied.