Genetic Information

Gene & Transcript Details

Gene
BRCA1
Transcript
NM_007294.4 MANE Select
Total Exons
Reference Sequence
NC_000017.10
Alternative Transcripts
IDStatusDetails
NM_007294.2 Alternative 7191 nt | 201–5792
NM_007294.3 RefSeq Select 7224 nt | 233–5824
NM_007294.4 MANE Select 7088 nt | 114–5705

Variant Details

HGVS Notation
NM_007294.4:c.4797A>G
Protein Change
A1599=
Location
Exon 15 (Exon 15 of )
15
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Likely Benign
1 publications
Clinical Statement

"This variant has been reported in ClinVar as Likely benign (3 clinical laboratories)."

COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene BRCA1.

Functional Studies & Therapeutic Relevance

Functional Summary

The variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Unknown
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 185 bp
- Donor Loss (DL) 0.0 30 bp
+ Acceptor Gain (AG) 0.0 121 bp
+ Donor Gain (DG) 0.01 -195 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines, PVS1 applies to null variants: "Very Strong Null variant (nonsense, frameshift, splice site (donor/acceptor +/-1,2), initiation codon, single or multi-exon deletion) in a gene where loss of function (LOF) is a known mechanism of disease." The evidence for this variant shows: NM_007294.4:c.4797A>G is a synonymous change, not predicted to abolish function or introduce a splice-site alteration. Therefore, this criterion is not applied because the variant is not a null variant.

PS1

PS1 (Not Applied)

According to VCEP guidelines, PS1 applies when a different nucleotide change results in the same amino acid change as a known pathogenic variant. The evidence shows: c.4797A>G is a synonymous change, not altering the amino acid. Therefore, PS1 is not applied.

PS2

PS2 (Not Applied)

According to standard ACMG guidelines, PS2 applies for de novo occurrence in a patient with confirmed parentage. No de novo data are available for this variant. Therefore, PS2 is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines, PS3 requires well-established functional studies supportive of a damaging effect. The evidence shows: no functional assays have been performed on c.4797A>G. Therefore, PS3 is not applied.

PS4

PS4 (Not Applied)

According to VCEP guidelines, PS4 applies when prevalence in affected individuals is significantly increased (OR ≥4, p≤0.05). The evidence shows: no case–control or aggregate clinical data demonstrating enrichment. Therefore, PS4 is not applied.

PM1

PM1 (Not Applied)

According to VCEP guidelines, PM1 applies to variants in established functional domains or mutational hotspots. The evidence shows: c.4797A>G is outside the BRCA1 RING, coiled-coil, and BRCT domains. Therefore, PM1 is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, PM2 Supporting: "Absent from controls in an outbred population, from gnomAD v2.1 (non-cancer) and gnomAD v3.1 (non-cancer)." The evidence shows: c.4797A>G is not present in gnomAD or other population databases. Therefore, PM2 is applied at Supporting strength because the variant is absent from controls.

PM3

PM3 (Not Applied)

According to VCEP guidelines, PM3 applies to recessive disorders with observed trans configuration. BRCA1 c.4797A>G is not in a recessive Fanconi anemia context. Therefore, PM3 is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, PM4 applies to protein length changes (in-frame indels). The evidence shows: c.4797A>G is synonymous with no effect on protein length. Therefore, PM4 is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines, PM5 applies to novel missense changes at codons with known pathogenic missense variants. c.4797A>G is synonymous, not missense. Therefore, PM5 is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, PM6 applies to presumed de novo cases without confirmation. No de novo information is available. Therefore, PM6 is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines, PP1 applies for co-segregation with disease in multiple affected family members. No segregation data are available for this variant. Therefore, PP1 is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, PP2 applies to missense variants in genes with low benign missense rate. c.4797A>G is synonymous. Therefore, PP2 is not applied.

PP3

PP3 (Not Applied)

According to VCEP guidelines, PP3 applies to computational predictions of deleterious effect (BayesDel ≥0.28 or SpliceAI ≥0.2). The evidence shows: SpliceAI score 0.01 (no splicing impact) and no protein change. Therefore, PP3 is not applied.

PP4

PP4 (Not Applied)

According to VCEP guidelines, PP4 applies to specific clinical phenotype data contributing to pathogenicity. No phenotype data specific to this variant are available. Therefore, PP4 is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, PP5 applies when a reputable source classifies as pathogenic without available evidence. ClinVar classifies as Likely Benign, not pathogenic. Therefore, PP5 is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, BA1 applies when allele frequency >0.1%. The evidence shows: c.4797A>G is absent from population databases, not exceeding threshold. Therefore, BA1 is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, BS1 applies when allele frequency >0.01%. The evidence shows: variant absent. Therefore, BS1 is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, BS2 applies in absence of recessive disease features (Fanconi anemia). No relevant homozygous or phenotypic data. Therefore, BS2 is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, BS3 requires well-established functional studies showing no damaging effect. No such studies exist for this variant. Therefore, BS3 is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines, BS4 applies for lack of segregation in affected family members. No segregation data are available. Therefore, BS4 is not applied.

BP1

BP1 (Not Applied)

According to VCEP guidelines, BP1 applies to missense variants outside functional domains. c.4797A>G is synonymous. Therefore, BP1 is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, BP2 applies when variant co-occurs in trans with a pathogenic variant in a dominant gene. No such data exist. Therefore, BP2 is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, BP3 applies to in-frame indels in repetitive regions. c.4797A>G is not an indel. Therefore, BP3 is not applied.

BP4

BP4 (Supporting)

According to VCEP guidelines, BP4 Supporting: "Missense or in-frame insertion, deletion or delins variants inside a (potentially) clinically important functional domain, and no predicted impact via protein change or splicing (BayesDel no-AF score ≤0.15 AND SpliceAI ≤0.1)." The evidence shows: SpliceAI predicts no impact on splicing (score 0.01) and no amino acid change. Therefore, BP4 is applied at Supporting strength because multiple computational tools predict no impact.

BP5

BP5 (Not Applied)

According to VCEP guidelines, BP5 applies to co-observation with pathogenic variants in other genes without phenotype. No such co-occurrence data. Therefore, BP5 is not applied.

BP6

BP6 (Supporting)

According to standard ACMG guidelines, BP6: "Reputable source recently reports variant as benign, but the evidence is not available for independent evaluation." The evidence shows: ClinVar lists this variant as Likely Benign from three clinical laboratories. Therefore, BP6 is applied at Supporting strength.

BP7

BP7 (Supporting)

According to standard ACMG guidelines, BP7: "A synonymous (silent) variant for which splicing prediction algorithms predict no impact to the splice consensus sequence nor the creation of a new splice site." The evidence shows: SpliceAI score 0.01, no splice impact. Therefore, BP7 is applied at Supporting strength.