Genetic Information

Gene & Transcript Details

Gene
BRCA2
Transcript
NM_000059.4 MANE Select
Total Exons
Reference Sequence
NC_000013.10
Alternative Transcripts
IDStatusDetails
NM_000059.4 MANE Select 11954 nt | 200–10456
NM_000059.2 Alternative 11386 nt | 228–10484
NM_000059.3 RefSeq Select 11386 nt | 228–10484

Variant Details

HGVS Notation
NM_000059.4:c.7765C>A
Protein Change
P2589T
Location
Exon 16 (Exon 16 of )
16
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.000398 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Uncertain Significance (VUS)
1 publications
Clinical Statement

"This variant has been reported in ClinVar as Uncertain significance (4 clinical laboratories)."

COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene BRCA2.

Functional Studies & Therapeutic Relevance

Functional Summary

The BRCA2 P2589T variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 -103 bp
- Donor Loss (DL) 0.0 40 bp
+ Acceptor Gain (AG) 0.0 -408 bp
+ Donor Gain (DG) 0.04 -60 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines for PVS1: "Very Strong Null variant (nonsense, frameshift, splice site ... loss of function) in a gene where LOF is a known mechanism of disease." The evidence shows this is a missense variant (P2589T), not a null variant. Therefore, PVS1 is not applied.

PS1

PS1 (Not Applied)

According to VCEP guidelines for PS1: "Strong Apply PS1 for predicted missense substitutions where a previously classified pathogenic variant has the same amino acid change." There is no previously established pathogenic variant with P2589T. Therefore, PS1 is not applied.

PS2

PS2 (Not Applied)

According to standard ACMG guidelines for PS2: "De novo (both maternity and paternity confirmed) in a patient with the disease and no family history." There is no evidence of de novo occurrence. Therefore, PS2 is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines for PS3: "Well-established in vitro or in vivo functional studies supportive of a damaging effect." No functional studies exist for this variant. Therefore, PS3 is not applied.

PS4

PS4 (Not Applied)

According to VCEP guidelines for PS4: "Prevalence in affected individuals is significantly increased compared to controls (OR ≥4, p ≤0.05)." No case-control data are available. Therefore, PS4 is not applied.

PM1

PM1 (Moderate)

According to standard ACMG guidelines for PM1: "Located in a mutational hot spot and/or critical functional domain without benign variation (Moderate)." The variant P2589T lies within the BRCA2 DNA binding domain (aa2481–3186), a critical functional region. Therefore, PM1 is applied at Moderate strength.

PM2

PM2 (Supporting)

According to VCEP guidelines for PM2: "Supporting Absent from controls in gnomAD v2.1 (non-cancer exome) and gnomAD v3.1 (non-cancer)." The variant is extremely rare (MAF = 0.000398%, 1/251418 alleles) and absent in non-cancer subset. Therefore, PM2 is applied at Supporting strength.

PM3

PM3 (Not Applied)

According to VCEP guidelines for PM3: "Apply for BRCA2-related Fanconi Anemia phenotype with co-occurrence of variants." No Fanconi Anemia phenotype or co-occurrence data are available. Therefore, PM3 is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines for PM4: "Protein length changes due to in-frame indels or stop-loss variants." This is a missense variant. Therefore, PM4 is not applied.

PM5

PM5 (Not Applied)

According to standard ACMG guidelines for PM5: "A different amino acid change at the same codon is pathogenic (Moderate)." No other pathogenic change at codon 2589 is reported. Therefore, PM5 is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines for PM6: "Assumed de novo without confirmation of paternity and maternity." No de novo data are available. Therefore, PM6 is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines for PP1: "Co-segregation with disease in multiple affected family members (Supporting to Strong)." No segregation data are available. Therefore, PP1 is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines for PP2: "Missense variant in a gene with a low rate of benign missense variation and pathogenic missense is common." BRCA2 has both benign and pathogenic missense variants; no clear evidence supports PP2. Therefore, PP2 is not applied.

PP3

PP3 (Not Applied)

According to VCEP guidelines for PP3: "Supporting predicted impact via protein change (BayesDel ≥0.30) or splicing (SpliceAI ≥0.2)." In silico predictions are mixed and SpliceAI = 0.04. Thus, PP3 is not applied.

PP4

PP4 (Not Applied)

According to VCEP guidelines for PP4: "Phenotype-based multifactorial likelihood data required for breast cancer." No such data are provided. Therefore, PP4 is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines for PP5: "Reputable source classifies variant as pathogenic." ClinVar reports this variant as VUS. Therefore, PP5 is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines for BA1: "Filter allele frequency >0.001 in gnomAD non-cancer indicates stand-alone benign." The variant frequency (0.00000398) is below this threshold. Therefore, BA1 is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines for BS1: "Filter allele frequency >0.0001 (Strong) or >0.00002 (Supporting) and ≤0.0001." The variant frequency (0.00000398) is below both thresholds. Therefore, BS1 is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines for BS2: "Observed in healthy adults without recessive disease phenotype (Fanconi Anemia)." No phenotype data on carriers are available. Therefore, BS2 is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines for BS3: "Well-established functional studies show no damaging effect." No such studies exist. Therefore, BS3 is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines for BS4: "Lack of segregation in affected family members." No segregation data are available. Therefore, BS4 is not applied.

BP1

BP1 (Not Applied)

According to VCEP guidelines for BP1: "Missense variant outside clinically important domain and no splicing impact." This variant lies inside the DNA binding domain. Therefore, BP1 is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines for BP2: "Observed in trans with a pathogenic variant for a dominant disorder." No co-occurrence data are available. Therefore, BP2 is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines for BP3: "In-frame indels in repetitive regions without known function." This is a missense variant. Therefore, BP3 is not applied.

BP4

BP4 (Supporting)

According to VCEP guidelines for BP4: "Supporting No predicted impact via protein change or splicing (BayesDel ≤0.18 and SpliceAI ≤0.1)." Computational evidence is mixed with SpliceAI = 0.04 and overall in silico favoring benign. Therefore, BP4 is applied at Supporting strength.

BP5

BP5 (Not Applied)

According to VCEP guidelines for BP5: "Cases with pathogenic variants in other breast-ovarian cancer genes without specific phenotype." No such co-observations are reported. Therefore, BP5 is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines for BP6: "Reputable source classifies variant as benign." ClinVar reports this variant as VUS. Therefore, BP6 is not applied.

BP7

BP7 (Not Applied)

According to VCEP guidelines for BP7: "Silent or intronic variants outside splice sites with no impact." This variant is missense. Therefore, BP7 is not applied.