Genetic Information

Gene & Transcript Details

Gene
PTEN
Transcript
NM_000314.8 MANE Select
Total Exons
Reference Sequence
NC_000010.10
Alternative Transcripts
IDStatusDetails
NM_000314.7 RefSeq Select 8514 nt | 845–2056
NM_000314.5 Alternative 8719 nt | 1032–2243
NM_000314.4 Alternative 5572 nt | 1032–2243
NM_000314.3 Alternative 3416 nt | 1032–2243
NM_000314.6 Alternative 8718 nt | 1032–2243
NM_000314.8 MANE Select 8515 nt | 846–2057

Variant Details

HGVS Notation
NM_000314.8:c.1027-10C>A
Protein Change
?
Location
Exon 8 (Exon 8 of )
8
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene PTEN.

Functional Studies & Therapeutic Relevance

Functional Summary

The variant PTEN 1027-10C>A has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.01 10 bp
- Donor Loss (DL) 0.0 -124 bp
+ Acceptor Gain (AG) 0.0 117 bp
+ Donor Gain (DG) 0.0 189 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines, the PVS1 rule for PTEN is determined by the PTEN PVS1 decision tree. The variant NM_000314.8:c.1027-10C>A lies outside the canonical ±1/±2 splice sites and there is no evidence of loss‐of‐function by splicing. Therefore, PVS1 is not applied because the variant is intronic beyond the critical splice positions with no predicted impact.

PS1

PS1 (Not Applied)

According to VCEP guidelines, PS1 requires the same amino acid change as a known pathogenic variant or an equivalent splicing impact. This variant is intronic with no coding or splicing match to a known pathogenic PTEN variant. Therefore, PS1 is not applied.

PS2

PS2 (Not Applied)

According to VCEP guidelines, PS2 requires confirmed de novo occurrence. No de novo data are available for this variant. Therefore, PS2 is not applied.

PS3

PS3 (Not Applied)

According to PTEN pre-processing, no functional studies (in vitro or in vivo) have been reported for NM_000314.8:c.1027-10C>A and the variant was not found in the PTEN functional assay file. Therefore, PS3 is not applied due to lack of functional evidence.

PS4

PS4 (Not Applied)

According to VCEP guidelines, PS4 requires increased prevalence in affected individuals or proband specificity scores. No case‐level or case‐control data are available for this variant. Therefore, PS4 is not applied.

PM1

PM1 (Not Applied)

According to VCEP guidelines, PM1 applies to variants in critical PTEN functional domains (residues 90-94, 123-130, 166-168). This intronic variant does not lie within a defined hotspot or functional domain. Therefore, PM1 is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, PM2 Supporting is 'Absent in population Databases present at <0.00001 allele frequency in gnomAD'. The variant is not found in gnomAD (MAF=0%). Therefore, PM2 is applied at Supporting strength because the variant is absent from large population databases.

PM3

PM3 (Not Applied)

According to VCEP guidelines, PM3 applies to recessive disorders observed in trans with a pathogenic variant. PTEN-associated conditions are dominant and no trans observations are available. Therefore, PM3 is not applied.

PM4

PM4 (Not Applied)

According to VCEP guidelines, PM4 applies to protein length changes (in-frame indels or stop-loss). This variant is intronic without protein length alteration. Therefore, PM4 is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines, PM5 requires a novel missense change at a residue with a known pathogenic missense. This is intronic. Therefore, PM5 is not applied.

PM6

PM6 (Not Applied)

According to VCEP guidelines, PM6 applies to presumed de novo cases. No de novo information is available. Therefore, PM6 is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines, PP1 requires co-segregation data. No segregation data are reported. Therefore, PP1 is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, PP2 applies to missense in genes with low benign missense rate. This is intronic. Therefore, PP2 is not applied.

PP3

PP3 (Not Applied)

According to VCEP guidelines, PP3 Supporting requires concordant computational evidence for deleterious effect (REVEL>0.7 or splicing algorithms). In silico predictors are mixed and SpliceAI=0.01 predicts no impact. Therefore, PP3 is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines, PP4 requires highly specific phenotype data. No phenotype data are available. Therefore, PP4 is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, PP5 requires a reputable source reporting pathogenicity. This variant is not in ClinVar or literature. Therefore, PP5 is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, BA1 Stand Alone requires gnomAD allele frequency >0.056%. The variant MAF=0%. Therefore, BA1 is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, BS1 Strong requires allele frequency 0.0043%–0.056%. The variant MAF=0%. Therefore, BS1 is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, BS2 applies to homozygous observations in healthy individuals. No such data exist. Therefore, BS2 is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, BS3 requires functional studies showing no damaging effect. No splicing or protein assays are available. Therefore, BS3 is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines, BS4 requires lack of segregation. No segregation studies exist. Therefore, BS4 is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, BP1 applies to missense variants in LOF genes. This is intronic. Therefore, BP1 is not applied.

BP2

BP2 (Not Applied)

According to VCEP guidelines, BP2 applies to trans observations with a pathogenic PTEN variant. No data are available. Therefore, BP2 is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, BP3 applies to in-frame indels in repeat regions. This is intronic. Therefore, BP3 is not applied.

BP4

BP4 (Supporting)

According to VCEP guidelines, BP4 Supporting is 'Multiple lines of computational evidence suggest no impact on gene or gene product.' SpliceAI=0.01 and CADD=2.28 predict no impact. Therefore, BP4 is applied at Supporting strength.

BP5

BP5 (Not Applied)

According to VCEP guidelines, BP5 applies when an alternate molecular basis explains disease. No alternate diagnosis data are available. Therefore, BP5 is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, BP6 requires a reputable source reporting benign impact. No such source exists. Therefore, BP6 is not applied.

BP7

BP7 (Supporting)

According to VCEP guidelines, BP7 Supporting is 'A synonymous or intronic variant at or beyond +7/-21 for which splicing prediction algorithms predict no impact.' This variant is at −10 (within the eligible region) and SpliceAI predicts no impact. Therefore, BP7 is applied at Supporting strength.