Genetic Information

Gene & Transcript Details

Gene
PTEN
Transcript
NM_000314.8 MANE Select
Total Exons
Reference Sequence
NC_000010.10
Alternative Transcripts
IDStatusDetails
NM_000314.7 RefSeq Select 8514 nt | 845–2056
NM_000314.5 Alternative 8719 nt | 1032–2243
NM_000314.4 Alternative 5572 nt | 1032–2243
NM_000314.3 Alternative 3416 nt | 1032–2243
NM_000314.6 Alternative 8718 nt | 1032–2243
NM_000314.8 MANE Select 8515 nt | 846–2057

Variant Details

HGVS Notation
NM_000314.8:c.745G>A
Protein Change
V249M
Location
Exon 7 (Exon 7 of )
7
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Uncertain Significance (VUS)
1 publications
Publications List
PMID: 29706350

The p.V249M variant (also known as c.745G>A), located in coding exon 7 of the PTEN gene, results from a G to A substitution at nucleotide position 745. The valine at codon 249 is replaced by methionine, an amino acid with highly similar properties. This variant demonstrated low intracellular protein abundance on one multiplex functional assay (Matreyek KA et al. Nat Genet, 2018 06;50:874-882) but had wildtype-like phosphatase activity in a massively parallel functional assay using a humanized yeast model (Mighell TL et al. Am J Hum Genet, 2018 05;102:943-955). This amino acid position is highly conserved in available vertebrate species. In addition, this alteration is predicted to be deleterious by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear.

Clinical Statement

"This variant has been reported in ClinVar as Uncertain significance (2 clinical laboratories)."

COSMIC Somatic Evidence

Open
COSMIC ID
COSM6837734
Recurrence
1 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene PTEN.

Functional Studies & Therapeutic Relevance

Functional Summary

The PTEN V249M variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.04 -56 bp
- Donor Loss (DL) 0.0 -208 bp
+ Acceptor Gain (AG) 0.0 -254 bp
+ Donor Gain (DG) 0.0 -29 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines, the PVS1 rule is: "Very Strong Strength: Very Strong Use PTEN PVS1 decision tree. Modification Type: Disease-specific". The variant is a missense change (c.745G>A; p.V249M) and not a null variant as defined by the PVS1 decision tree. Therefore, this criterion is not applied.

PS1

PS1 (Not Applied)

According to VCEP guidelines, the PS1 rule is: "Strong Strength: Strong Same amino acid change as a previously established pathogenic variant regardless of nucleotide change OR different variant at same nucleotide position as a pathogenic splicing variant, where in silico models predict impact equal to or greater than the known pathogenic variant." There is no known pathogenic variant at codon V249 producing the same amino acid change. Therefore, PS1 is not applied.

PS2

PS2 (Not Applied)

According to VCEP guidelines, the PS2 rule is: "Strong Strength: Strong De novo (both maternity and paternity confirmed) observation in a patient with the disease and no family history." No de novo or parental testing data are available for this variant. Therefore, PS2 is not applied.

PS3

PS3 (Not Applied)

According to PTEN Pre-processing findings, the PS3_Moderate threshold is -1.11, and the variant's phosphatase activity score is -0.3069, which did not meet the threshold. Therefore, PS3 is not applied.

PS4

PS4 (Not Applied)

According to VCEP guidelines, the PS4 rule is: "Strong Strength: Strong Probands with specificity score 4-15.5 (see text) OR The prevalence of the variant in affected individuals is significantly increased compared with the prevalence in controls." No case or cohort data are available to calculate specificity scores or prevalence. Therefore, PS4 is not applied.

PM1

PM1 (Not Applied)

According to VCEP guidelines, the PM1 rule is: "Moderate Strength: Moderate Located in a mutational hot spot and/or critical and well-established functional domain. Defined to include residues in catalytic motifs: 90-94, 123-130, 166-168 (NP_000305.3)." The V249 residue is outside these defined catalytic motifs. Therefore, PM1 is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, the PM2 rule is: "Supporting Strength: Supporting Absent in population Databases present at <0.00001 (0.001%) allele frequency in gnomAD or another large sequenced population. If multiple alleles are present within any subpopulation, allele frequency in that subpopulation must be <0.00002 (0.002%)." This variant is absent from gnomAD and other population databases. Therefore, PM2 is applied at Supporting strength.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines, the PM3 rule is: "For recessive disorders, detected in trans with a pathogenic variant." PTEN-related disorders are autosomal dominant and there is no evidence of trans configuration. Therefore, PM3 is not applied.

PM4

PM4 (Not Applied)

According to VCEP guidelines, the PM4 rule is: "Moderate Strength: Moderate Protein length changes due to in-frame deletions/insertions in a non-repeat region or stop-loss variants. Applies to in-frame insertions or deletions impacting at least one residue in a catalytic motif, and variants causing protein extension." This variant is a single amino acid substitution, not an in-frame indel or protein-length change. Therefore, PM4 is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines, the PM5 rule is: "Moderate Strength: Moderate Missense change at an amino acid residue where a different missense change determined to be pathogenic or likely pathogenic has been seen before, with BLOSUM62 score equal to or less than the known variant." No other pathogenic missense variants at residue V249 have been reported. Therefore, PM5 is not applied.

PM6

PM6 (Not Applied)

According to VCEP guidelines, the PM6 rule is: "Strong/Very Strong Strength: Assumed de novo observations without confirmation of paternity/maternity in a patient with the disease and no family history." No presumed de novo data exist. Therefore, PM6 is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines, the PP1 rule is: "Supporting/Moderate/Strong Strength: Co-segregation with disease in multiple affected family members with varying numbers of meioses observed." No family segregation data are available. Therefore, PP1 is not applied.

PP2

PP2 (Supporting)

According to standard ACMG guidelines, the PP2 rule is: "Supporting Strength: Supporting Missense variant in a gene that has a low rate of benign missense variation and where missense variants are a common mechanism of disease." PTEN is intolerant to benign missense variation and missense changes are a well-established mechanism in PTEN-related disease. Therefore, PP2 is applied at Supporting strength.

PP3

PP3 (Not Applied)

According to VCEP guidelines, the PP3 rule is: "Supporting Strength: Supporting Multiple lines of computational evidence support a deleterious effect on the gene or gene product. Missense variants: REVEL score > 0.7." Computational predictions for this variant are mixed and no consensus REVEL score > 0.7 is available. Therefore, PP3 is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines, the PP4 rule is: "Supporting Strength: Supporting Patient's phenotype or family history is highly specific for a disease with a single genetic etiology." No clinical phenotype or family history data are provided. Therefore, PP4 is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, the PP5 rule is: "Supporting Strength: Supporting A reputable source reports the variant as pathogenic." ClinVar reports this variant as Uncertain Significance. Therefore, PP5 is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, the BA1 rule is: "Stand Alone Strength: Stand Alone gnomAD Filtering allele frequency >0.00056 (0.056%)." The variant is absent from population databases. Therefore, BA1 is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, the BS1 rule is: "Strong Strength: Strong gnomAD Filtering allele frequency from 0.000043 (0.0043%) up to 0.00056 (0.056%)." The variant is absent from population databases. Therefore, BS1 is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, the BS2 rule is: "Strong Strength: Strong Observed in the homozygous state in a healthy or PHTS-unaffected individual." No homozygous observations in healthy individuals are reported. Therefore, BS2 is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, the BS3 rule is: "Strong Strength: Strong Well-established in vitro or in vivo functional studies shows no damaging effect on protein function." Functional assay data do not demonstrate normal function (phosphatase activity score is -0.3069, not >0). Therefore, BS3 is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines, the BS4 rule is: "Strong Strength: Strong Lack of segregation in affected members of two or more families." No segregation data are available. Therefore, BS4 is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, the BP1 rule is: "Supporting Strength: Supporting Missense variant in a gene for which primarily truncating variants cause disease." Missense variants in PTEN are a known mechanism of disease. Therefore, BP1 is not applied.

BP2

BP2 (Not Applied)

According to VCEP guidelines, the BP2 rule is: "Supporting Strength: Supporting Observed in trans with a pathogenic or likely pathogenic PTEN variant OR at least three observations in cis with different pathogenic/likely pathogenic PTEN variants." No cis/trans data with other PTEN variants are available. Therefore, BP2 is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, the BP3 rule is: "Supporting Strength: Supporting In-frame deletions/insertions in a repetitive region without a known function." This is a missense substitution, not an indel. Therefore, BP3 is not applied.

BP4

BP4 (Not Applied)

According to VCEP guidelines, the BP4 rule is: "Supporting Strength: Supporting Multiple lines of computational evidence suggest no impact on gene or gene product. Missense variants: REVEL scores < 0.5." Computational evidence is mixed and no consensus REVEL < 0.5 is available. Therefore, BP4 is not applied.

BP5

BP5 (Not Applied)

According to VCEP guidelines, the BP5 rule is: "Supporting Strength: Supporting Variant found in a case with an alternate molecular basis for disease." No such alternate molecular basis data are provided. Therefore, BP5 is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, the BP6 rule is: "Supporting Strength: Supporting A reputable source reports the variant as benign." No reputable source lists this variant as benign. Therefore, BP6 is not applied.

BP7

BP7 (Not Applied)

According to standard ACMG guidelines, the BP7 rule is: "Supporting Strength: Supporting A synonymous or intronic variant at or beyond +7/-21 with no splicing impact." This is a missense variant, not synonymous or intronic. Therefore, BP7 is not applied.