Genetic Information

Gene & Transcript Details

Gene
ASXL1
Transcript
NM_015338.5 MANE Select
Total Exons
Reference Sequence
NC_000020.10
Alternative Transcripts
IDStatusDetails
NM_015338.5 RefSeq Select 7056 nt | 433–5058
NM_015338.4 Alternative 7047 nt | 427–5052
NM_015338.3 Alternative 7026 nt | 420–5045
NM_015338.6 MANE Select 7052 nt | 446–5071

Variant Details

HGVS Notation
NM_015338.5:c.1954G>A
Protein Change
G652S
Location
Exon 13 (Exon 13 of )
13
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
1.78 in 100,000
Extremely Rare
ACMG Criteria Applied BA1

ClinVar

Open
Classification
Unknown (No data to parse)
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
COSM1716555
Recurrence
14 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene ASXL1.

Functional Studies & Therapeutic Relevance

Functional Summary

The ASXL1 G652S variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 -234 bp
- Donor Loss (DL) 0.0 411 bp
+ Acceptor Gain (AG) 0.0 -84 bp
+ Donor Gain (DG) 0.0 -85 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to standard ACMG guidelines, the rule for PVS1 is: "Null variant in a gene where loss of function (LoF) is a known mechanism of disease". The evidence for this variant shows it is a missense change (G652S) and not predicted to cause LoF. Therefore, this criterion is not applied.

PS1

PS1 (Not Applied)

According to standard ACMG guidelines, the rule for PS1 is: "Same amino acid change as a previously established pathogenic variant regardless of nucleotide change". There is no record of a known pathogenic variant causing G652S at this residue. Therefore, this criterion is not applied.

PS2

PS2 (Not Applied)

According to standard ACMG guidelines, the rule for PS2 is: "De novo (both maternity and paternity confirmed) in a patient with the disease and no family history". No de novo evidence is available. Therefore, this criterion is not applied.

PS3

PS3 (Not Applied)

According to standard ACMG guidelines, the rule for PS3 is: "Well-established functional studies supportive of a damaging effect on the gene or gene product". Functional studies for ASXL1 G652S are not available. Therefore, this criterion is not applied.

PS4

PS4 (Not Applied)

According to standard ACMG guidelines, the rule for PS4 is: "Prevalence in affected individuals significantly increased compared with controls". No case-control data are available. Therefore, this criterion is not applied.

PM1

PM1 (Not Applied)

According to standard ACMG guidelines, the rule for PM1 is: "Located in a mutational hot spot or well-established functional domain without benign variation". There is no evidence that G652S lies in such a domain. Therefore, this criterion is not applied.

PM2

PM2 (Not Applied)

According to standard ACMG guidelines, the rule for PM2 is: "Absent from controls (or at extremely low frequency if recessive)". The variant is present in gnomAD with MAF=1.78% overall and 8.02% in Admixed American. Therefore, this criterion is not applied.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines, the rule for PM3 is: "Detected in trans with a pathogenic variant (for recessive disorders)". No evidence for recessive trans configuration is available. Therefore, this criterion is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, the rule for PM4 is: "Protein length changes due to in-frame deletions/insertions or stop-loss variants". This is a missense substitution without length change. Therefore, this criterion is not applied.

PM5

PM5 (Not Applied)

According to standard ACMG guidelines, the rule for PM5 is: "Novel missense change at an amino acid residue where a different pathogenic missense change has been seen". No other pathogenic missense changes at residue 652 are reported. Therefore, this criterion is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, the rule for PM6 is: "Assumed de novo, but without confirmation of paternity and maternity". No such evidence is available. Therefore, this criterion is not applied.

PP1

PP1 (Not Applied)

According to standard ACMG guidelines, the rule for PP1 is: "Co-segregation with disease in multiple affected family members". No segregation data are available. Therefore, this criterion is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, the rule for PP2 is: "Missense variant in a gene with a low rate of benign missense variation and where missense variants are a common mechanism of disease". ASXL1 disease mechanism is loss-of-function, not missense, and the gene has observed missense variation. Therefore, this criterion is not applied.

PP3

PP3 (Not Applied)

According to standard ACMG guidelines, the rule for PP3 is: "Multiple lines of computational evidence support a deleterious effect". In silico predictions are predominantly benign and SpliceAI predicts no impact on splicing. Therefore, this criterion is not applied.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines, the rule for PP4 is: "Patient's phenotype or family history highly specific for a disease with a single genetic etiology". No phenotype information is provided. Therefore, this criterion is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, the rule for PP5 is: "Reputable source reports variant as pathogenic, but without accessible evidence". No such reports exist. Therefore, this criterion is not applied.

BA1

BA1 (Stand Alone)

According to standard ACMG guidelines, the rule for BA1 is: "Allele frequency is too high for the disorder (based on population data)". The variant has MAF=8.02% in the Admixed American population, exceeding the 5% BA1 threshold. Therefore, this criterion is applied at Stand Alone strength because allele frequency far exceeds the threshold.

BS1

BS1 (Strong)

According to standard ACMG guidelines, the rule for BS1 is: "Allele frequency is greater than expected for disorder". The variant's overall MAF (1.78%) and subpopulation MAF (8.02%) are much higher than expected for a rare dominant disorder. Therefore, this criterion is applied at Strong strength.

BS2

BS2 (Strong)

According to standard ACMG guidelines, the rule for BS2 is: "Observed in a healthy adult individual for a dominant disorder with full penetrance expected at an early age". There are 176 homozygotes and thousands of carriers in gnomAD without reported disease. Therefore, this criterion is applied at Strong strength.

BS3

BS3 (Not Applied)

According to standard ACMG guidelines, the rule for BS3 is: "Well-established functional studies show no damaging effect on protein function or splicing". No such functional assays are available. Therefore, this criterion is not applied.

BS4

BS4 (Not Applied)

According to standard ACMG guidelines, the rule for BS4 is: "Lack of segregation in affected family members". No family segregation data are available. Therefore, this criterion is not applied.

BP1

BP1 (Supporting)

According to standard ACMG guidelines, the rule for BP1 is: "Missense variant in a gene where only LoF causes disease". ASXL1 disease is associated with loss-of-function variants, not missense. Therefore, this criterion is applied at Supporting strength.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, the rule for BP2 is: "Observed in trans with a pathogenic variant for dominant disorders or in cis with a pathogenic variant". No such observations are reported. Therefore, this criterion is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, the rule for BP3 is: "In-frame deletions/insertions in a repetitive region without known function". This is a missense substitution. Therefore, this criterion is not applied.

BP4

BP4 (Supporting)

According to standard ACMG guidelines, the rule for BP4 is: "Multiple lines of computational evidence suggest no impact on gene or gene product". In silico tools (CADD, PolyPhen-2, MetaSVM, MetaLR, PrimateAI) predict benign impact and SpliceAI predicts no splicing effect. Therefore, this criterion is applied at Supporting strength.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines, the rule for BP5 is: "Variant found in a case with an alternate molecular basis for disease". No such case reports are available. Therefore, this criterion is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, the rule for BP6 is: "Reputable source reports variant as benign, but without accessible evidence". No such reports are available. Therefore, this criterion is not applied.

BP7

BP7 (Not Applied)

According to standard ACMG guidelines, the rule for BP7 is: "Synonymous variant with no predicted impact on splicing". This is a missense variant, not synonymous. Therefore, this criterion is not applied.