Genetic Information

Gene & Transcript Details

Gene
TP53
Transcript
NM_000546.5 MANE Select
Total Exons
Reference Sequence
NC_000017.10
Alternative Transcripts
IDStatusDetails
NM_000546.3 Alternative 2640 nt | 252–1433
NM_000546.5 RefSeq Select 2591 nt | 203–1384
NM_000546.6 MANE Select 2512 nt | 143–1324
NM_000546.4 Alternative 2586 nt | 198–1379
NM_000546.2 Alternative 2629 nt | 252–1433

Variant Details

HGVS Notation
NM_000546.5:c.469G>T
Protein Change
V157F
Location
Exon 5 (Exon 5 of )
5
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
COSM10670
Recurrence
355 occurrences
PM1 Criteria
Applied
COSMIC Database Preview
COSMIC Preview
Accessing full COSMIC database details requires institutional login or subscription.

Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene TP53.

Functional Studies & Therapeutic Relevance

Functional Summary

The TP53 V157F variant has been functionally characterized and is likely damaging. It is located in the DNA-binding domain of the TP53 protein. In vitro and structural studies have shown that this mutation results in decreased DNA-binding activity and impaired transcriptional activity. The mutant protein exhibits a rearranged DNA-binding surface, reduced expression of pro-apoptotic proteins, and a diminished apoptotic response. Additionally, the variant confers a gain of function, leading to aberrant gene regulation in cell culture.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 -143 bp
- Donor Loss (DL) 0.0 -44 bp
+ Acceptor Gain (AG) 0.0 -189 bp
+ Donor Gain (DG) 0.0 35 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines, PVS1 applies only to null variants resulting in loss of function via nonsense, frameshift, canonical splice, or initiation codon disruptions. The variant NM_000546.5:c.469G>T (p.V157F) is a missense change. Therefore, PVS1 is not applied because this is not a null variant.

PS1

PS1 (Not Applied)

According to VCEP guidelines, PS1 applies when the variant results in an amino acid change previously established as pathogenic at the same residue. There is no report of a different nucleotide change causing p.V157F or any other pathogenic variant at codon 157. Therefore, PS1 is not applied because there is no identical amino acid change asserted as pathogenic.

PS2

PS2 (Not Applied)

According to VCEP guidelines, PS2 applies to confirmed de novo occurrences with specific point thresholds. No de novo data are available for this variant. Therefore, PS2 is not applied due to lack of de novo evidence.

PS3

PS3 (Strong)

According to VCEP guidelines, PS3_Strong: "Non-functional on Kato et al. data AND loss of function (LOF) on another assay." The evidence for p.V157F shows well-established in vitro and structural studies demonstrating decreased DNA-binding activity, impaired transcriptional function, reduced pro-apoptotic protein expression, and a gain-of-function oncogenic phenotype. Therefore, PS3 is applied at Strong strength because multiple functional assays indicate non-function and LOF consistent with the VCEP PS3_Strong rule.

PS4

PS4 (Not Applied)

According to VCEP guidelines, PS4 applies based on proband counting and case–control data with point thresholds. No case or proband data meeting VCEP thresholds were provided. Therefore, PS4 is not applied due to absence of qualifying clinical case evidence.

PM1

PM1 (Not Applied)

According to VCEP guidelines, PM1 applies to missense variants within TP53 hotspot codons (175, 245, 248, 249, 273, 282). Codon 157 is outside these specified hotspots. Therefore, PM1 is not applied because the variant is not located in a VCEP-defined hotspot codon.

PM2

PM2 (Supporting)

According to VCEP guidelines, PM2_Supporting: "Allele frequency <0.00003 in gnomAD or large population databases." The variant p.V157F is absent from gnomAD and other population databases. Therefore, PM2 is applied at Supporting strength because the allele frequency meets the VCEP PM2_Supporting threshold.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines, PM3 applies to variants observed in trans with a pathogenic variant in recessive disorders. TP53 disorders are dominant and no trans observations are relevant. Therefore, PM3 is not applied because the variant is not in a recessive context.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, PM4 applies to protein length–altering variants such as in-frame indels or stop‐loss variants. The variant p.V157F is a missense change without alteration of protein length. Therefore, PM4 is not applied.

PM5

PM5 (Not Applied)

According to VCEP guidelines, PM5 applies when ≥1 different missense variant at the same residue has been classified as (Likely) Pathogenic. There are no other reported pathogenic or likely pathogenic missense variants at codon 157. Therefore, PM5 is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, PM6 applies to assumed de novo occurrences without confirmation of paternity/maternity. No such data are available for this variant. Therefore, PM6 is not applied.

PP1

PP1 (Not Applied)

According to VCEP guidelines, PP1 applies for co-segregation with disease in families meeting specific meiosis thresholds. No segregation data are available. Therefore, PP1 is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, PP2 applies when a gene has low rate of benign missense variation and missense is common mechanism. TP53 has both benign and pathogenic missense, and no VCEP specification uses PP2. Therefore, PP2 is not applied.

PP3

PP3 (Not Applied)

According to VCEP guidelines, PP3_Moderate requires BayesDel ≥0.16 and no predicted splicing impact. BayesDel data are not provided and in silico results are mixed. Therefore, PP3 is not applied.

PP4

PP4 (Not Applied)

According to VCEP guidelines, PP4 applies for LFS-associated tumor types with specific VAF criteria. No tumor VAF or phenotype data are provided. Therefore, PP4 is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, PP5 applies for reputable source assertions without available data. No reputable source assertions (e.g., ClinVar) are present. Therefore, PP5 is not applied.

BA1

BA1 (Not Applied)

According to VCEP guidelines, BA1 requires allele frequency ≥0.001 in gnomAD subpopulations. The variant is absent from population databases. Therefore, BA1 is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, BS1 requires filtering allele frequency ≥0.0003 but <0.001. The variant is absent from gnomAD. Therefore, BS1 is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, BS2 requires observation in ≥2 unrelated older unaffected females. No such data are available. Therefore, BS2 is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, BS3 applies when functional assays show no loss of function. The functional data for p.V157F demonstrate damaging effects. Therefore, BS3 is not applied.

BS4

BS4 (Not Applied)

According to VCEP guidelines, BS4 applies when there is lack of co-segregation in affected family members. No segregation data are available. Therefore, BS4 is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, BP1 applies to missense variants in genes where only truncating variants cause disease. TP53 has pathogenic missense variants, so BP1 is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, BP2 applies when observed in cis with a pathogenic variant in dominant disorders. No such observations are reported. Therefore, BP2 is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, BP3 applies to in‐frame indels in repetitive regions. This is a missense variant, so BP3 is not applied.

BP4

BP4 (Supporting)

According to standard ACMG guidelines, BP4: "Multiple lines of computational evidence suggest no impact on gene or gene product." SpliceAI predicts no splicing impact and CADD score is low (2.39), though other in silico tools are mixed. Therefore, BP4 is applied at Supporting strength because overall computational evidence leans benign for splicing and functional impact.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines, BP5 applies when a variant is found in a case with an alternate molecular cause. No alternate cause is reported. Therefore, BP5 is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, BP6 applies to unpublished reputable source assertions without data. No such assertions exist. Therefore, BP6 is not applied.

BP7

BP7 (Not Applied)

According to VCEP guidelines, BP7 applies to synonymous/intronic variants with no splicing impact. This is a missense variant. Therefore, BP7 is not applied.