Genetic Information

Gene & Transcript Details

Gene
MSH2
Transcript
NM_000251.3 MANE Select
Total Exons
Reference Sequence
NC_000002.11
Alternative Transcripts
IDStatusDetails
NM_000251.3 MANE Select 3115 nt | 37–2841
NM_000251.1 Alternative 3145 nt | 69–2873
NM_000251.2 RefSeq Select 3226 nt | 126–2930

Variant Details

HGVS Notation
NM_000251.3:c.793-11_794dup
Protein Change
?
Location
Exon 4 (Exon 4 of )
4
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

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COSMIC Somatic Evidence

Open
COSMIC ID
Recurrence
0 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
Accessing full COSMIC database details requires institutional login or subscription.

Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene MSH2.

Functional Studies & Therapeutic Relevance

Functional Summary

The MSH2 793-11_794dup variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Unknown
PP3 Applied
Yes
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.01 13 bp
- Donor Loss (DL) 0.0 36 bp
+ Acceptor Gain (AG) 0.71 0 bp
+ Donor Gain (DG) 0.0 -227 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to VCEP guidelines, PVS1 applies to variants at the canonical splice positions (IVS±1 or IVS±2) or causing a null effect by nonsense/frameshift. This variant is at c.793-11, a non-canonical splice position and does not introduce a premature stop. Therefore, PVS1 is not applied.

PS1

PS1 (Not Applied)

According to standard ACMG guidelines, PS1 applies when a variant results in the same amino acid change as a known pathogenic variant. This intronic duplication does not encode a missense change. Therefore, PS1 is not applied.

PS2

PS2 (Not Applied)

According to standard ACMG guidelines, PS2 requires confirmed de novo occurrence with parental testing. No de novo data are available for this variant. Therefore, PS2 is not applied.

PS3

PS3 (Not Applied)

According to VCEP guidelines, PS3 requires validated functional assay evidence demonstrating a damaging effect. No functional studies have been performed for this variant. Therefore, PS3 is not applied.

PS4

PS4 (Not Applied)

According to standard ACMG guidelines, PS4 requires statistically significant case-control or segregation data. No such data are available. Therefore, PS4 is not applied.

PM1

PM1 (Not Applied)

According to standard ACMG guidelines, PM1 applies to variants in mutational hot spots or critical functional domains. This variant lies outside known hotspots/domains. Therefore, PM1 is not applied.

PM2

PM2 (Supporting)

According to VCEP guidelines, PM2_Supporting applies to variants absent or extremely rare (<1 in 50,000 alleles) in gnomAD v4. This variant is absent from gnomAD (MAF=0). Therefore, PM2 is applied at Supporting strength because it is absent from control databases.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines, PM3 applies to recessive disorders when a variant is observed in trans with a pathogenic variant. Lynch syndrome is dominant, and no trans data are available. Therefore, PM3 is not applied.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, PM4 applies to protein length changes due to in-frame indels. The consequence of this intronic duplication on the protein is unknown. Therefore, PM4 is not applied.

PM5

PM5 (Not Applied)

According to standard ACMG guidelines, PM5 applies to novel missense changes at residues where a different pathogenic missense change is established. This is an intronic duplication. Therefore, PM5 is not applied.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, PM6 applies to assumed de novo variants without parental confirmation. No de novo evidence is available. Therefore, PM6 is not applied.

PP1

PP1 (Not Applied)

According to standard ACMG guidelines, PP1 requires segregation data in affected family members. No segregation data are available. Therefore, PP1 is not applied.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, PP2 applies to missense variants in genes with low benign missense variation. This is an intronic variant. Therefore, PP2 is not applied.

PP3

PP3 (Supporting)

According to VCEP guidelines, PP3_Supporting applies for predicted splice defects at non-canonical sites using SpliceAI with a delta score ≥0.2. SpliceAI predicts an acceptor gain with a score of 0.71. Therefore, PP3 is applied at Supporting strength because in silico tools predict a moderate splicing impact.

PP4

PP4 (Not Applied)

According to VCEP guidelines, PP4 requires tumor phenotype data (e.g., MSI-H or loss of MMR expression) consistent with the variant. No tumor data are available. Therefore, PP4 is not applied.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, PP5 applies to assertions from reputable databases without evidence. No such assertions exist. Therefore, PP5 is not applied.

BA1

BA1 (Not Applied)

According to standard ACMG guidelines, BA1 requires allele frequency ≥0.1% in controls. This variant has MAF=0. Therefore, BA1 is not applied.

BS1

BS1 (Not Applied)

According to VCEP guidelines, BS1 requires allele frequency ≥0.01% and <0.1%. This variant has MAF=0. Therefore, BS1 is not applied.

BS2

BS2 (Not Applied)

According to VCEP guidelines, BS2 requires observation in trans with a pathogenic variant in unaffected individuals. No co-occurrence data are available. Therefore, BS2 is not applied.

BS3

BS3 (Not Applied)

According to VCEP guidelines, BS3 requires functional assays demonstrating no impact. No functional data exist. Therefore, BS3 is not applied.

BS4

BS4 (Not Applied)

According to standard ACMG guidelines, BS4 requires lack of segregation in multiple affected family members. No segregation data exist. Therefore, BS4 is not applied.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, BP1 applies to missense variants in genes where only truncating variants cause disease. This variant is intronic. Therefore, BP1 is not applied.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, BP2 applies when a variant is observed in cis with a pathogenic variant. No such data are available. Therefore, BP2 is not applied.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, BP3 applies to in-frame indels in repetitive regions. This variant is an intronic duplication; its repeat context is unknown. Therefore, BP3 is not applied.

BP4

BP4 (Not Applied)

According to VCEP guidelines, BP4 applies when SpliceAI predicts no splicing impact (delta ≤0.1). SpliceAI score is 0.71 indicating impact. Therefore, BP4 is not applied.

BP5

BP5 (Not Applied)

According to VCEP guidelines, BP5 requires tumor phenotypes inconsistent with the gene. No tumor data are available. Therefore, BP5 is not applied.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, BP6 requires a benign assertion from a reputable source. No such assertion exists. Therefore, BP6 is not applied.

BP7

BP7 (Not Applied)

According to VCEP guidelines, BP7 applies to intronic variants at or beyond –21 from the exon. This variant is at –11, within the splice region. Therefore, BP7 is not applied.