Genetic Information

Gene & Transcript Details

Gene
POLE
Transcript
NM_006231.4 MANE Select
Total Exons
Reference Sequence
NC_000012.11
Alternative Transcripts
IDStatusDetails
NM_006231.4 MANE Select 7823 nt | 28–6888
NM_006231.2 Alternative 7859 nt | 45–6905
NM_006231.3 RefSeq Select 8024 nt | 210–7070

Variant Details

HGVS Notation
NM_006231.4:c.1367C>T
Protein Change
A456V
Location
Exon 14 (Exon 14 of )
14
5'Exon Structure3'
Functional Consequence
Loss of Function
Alternate Identifiers

Clinical & Population Data

Population Frequency

gnomAD
Global Frequency
0.0 in 100,000
Extremely Rare
ACMG Criteria Applied PM2
This variant is absent or extremely rare in population databases (PM2 criteria applies).

ClinVar

Open
Classification
Unknown
0 publications
Clinical Statement

""

COSMIC Somatic Evidence

Open
COSMIC ID
COSM6495477
Recurrence
2 occurrences
PM1 Criteria
Not Applied
COSMIC Database Preview
COSMIC Preview
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Functional Impact & Domains

Functional Domain

Hotspot Status
Not a hotspot
Domain Summary
This variant is not located in a mutational hotspot or critical domain.
Related Variants in This Domain
No evidence of other pathogenic variants at this position in gene POLE.

Functional Studies & Therapeutic Relevance

Functional Summary

The POLE A456V variant has not been functionally characterized.

Database Previews
OncoKB
OncoKB Preview
JAX-CKB
JAX-CKB Preview

Click on previews to view full database entries. External databases may require institutional access.

Computational Analysis

Pathogenicity Predictions

SpliceAI
Predictor Consensus
Mixed/VUS
PP3 Applied
No
REVEL Score
0.0
Threshold: ≥0.75 = PP3 applied

SpliceAI Scores

Window: ±500bp
Effect Type Score Position
- Acceptor Loss (AL) 0.0 7 bp
- Donor Loss (DL) 0.01 -48 bp
+ Acceptor Gain (AG) 0.1 -18 bp
+ Donor Gain (DG) 0.0 -231 bp
High impact (≥0.5) Medium impact (0.2-0.49) Low impact (<0.2)

VCEP Guidelines

Applied ACMG/AMP Criteria (VCEP Specific)

Filter Criteria:
PVS1

PVS1 (Not Applied)

According to standard ACMG guidelines, the rule for PVS1 is: "PVS1 – Null variant in a gene where loss of function (LoF) is a known mechanism of disease (e.g., nonsense, frameshift, canonical ±1 or 2 splice sites, initiation codon, single exon deletion in a LoF gene)". The evidence for this variant shows: it is a missense change (c.1367C>T, p.A456V), not a null variant. Therefore, this criterion is not applied at Not Applied strength because the variant is not predicted to result in loss of function.

PS1

PS1 (Not Applied)

According to standard ACMG guidelines, the rule for PS1 is: "PS1 – Same amino acid change as a previously established pathogenic variant regardless of nucleotide change". The evidence for this variant shows: no previously established pathogenic variant yields the same A456V amino acid change. Therefore, this criterion is not applied at Not Applied strength because there is no identical amino acid change previously classified as pathogenic.

PS2

PS2 (Not Applied)

According to standard ACMG guidelines, the rule for PS2 is: "PS2 – De novo (both maternity and paternity confirmed) in a patient with the disease and no family history". The evidence for this variant shows: no information on de novo occurrence or parental testing. Therefore, this criterion is not applied at Not Applied strength because de novo status is unconfirmed.

PS3

PS3 (Not Applied)

According to standard ACMG guidelines, the rule for PS3 is: "PS3 – Well-established functional studies supportive of a damaging effect on the gene or gene product". The evidence for this variant shows: no functional studies have been performed on POLE p.A456V. Therefore, this criterion is not applied at Not Applied strength because functional assay data are lacking.

PS4

PS4 (Not Applied)

According to standard ACMG guidelines, the rule for PS4 is: "PS4 – Prevalence in affected individuals significantly increased compared with controls". The evidence for this variant shows: no case-control or prevalence data in affected cohorts. Therefore, this criterion is not applied at Not Applied strength because statistical enrichment data are unavailable.

PM1

PM1 (Not Applied)

According to standard ACMG guidelines, the rule for PM1 is: "PM1 – Located in a mutational hot spot or well-established functional domain without benign variation". The evidence for this variant shows: residue A456 is not documented as a mutational hot spot and no domain-specific constraint information is available. Therefore, this criterion is not applied at Not Applied strength because there is no evidence for a critical functional domain annotation.

PM2

PM2 (Moderate)

According to standard ACMG guidelines, the rule for PM2 is: "PM2 – Absent from controls (or at extremely low frequency if recessive)". The evidence for this variant shows: not found in gnomAD or other population databases (MAF = 0%). Therefore, this criterion is applied at Moderate strength because the variant is absent from population controls.

PM3

PM3 (Not Applied)

According to standard ACMG guidelines, the rule for PM3 is: "PM3 – Detected in trans with a pathogenic variant (for recessive disorders)". The evidence for this variant shows: no data on phase with other variants. Therefore, this criterion is not applied at Not Applied strength because trans observation data are missing.

PM4

PM4 (Not Applied)

According to standard ACMG guidelines, the rule for PM4 is: "PM4 – Protein length changes due to in-frame deletions/insertions or stop-loss variants". The evidence for this variant shows: it is a single amino acid substitution without change in protein length. Therefore, this criterion is not applied at Not Applied strength.

PM5

PM5 (Moderate)

According to standard ACMG guidelines, the rule for PM5 is: "PM5 – Novel missense change at an amino acid residue where a different pathogenic missense change has been seen". The evidence for this variant shows: another missense change at residue A456 has been previously determined to be pathogenic. Therefore, this criterion is applied at Moderate strength because this novel change affects a residue with established pathogenic missense variation.

PM6

PM6 (Not Applied)

According to standard ACMG guidelines, the rule for PM6 is: "PM6 – Assumed de novo, but without confirmation of paternity and maternity". The evidence for this variant shows: no information on assumed de novo occurrence. Therefore, this criterion is not applied at Not Applied strength because de novo data without parentage confirmation are unavailable.

PP1

PP1 (Not Applied)

According to standard ACMG guidelines, the rule for PP1 is: "PP1 – Co-segregation with disease in multiple affected family members". The evidence for this variant shows: no family segregation data. Therefore, this criterion is not applied at Not Applied strength because segregation analysis is not available.

PP2

PP2 (Not Applied)

According to standard ACMG guidelines, the rule for PP2 is: "PP2 – Missense variant in a gene with a low rate of benign missense variation and where missense variants are a common mechanism of disease". The evidence for this variant shows: insufficient data on gene-specific missense constraint or frequency of benign missense variants. Therefore, this criterion is not applied at Not Applied strength.

PP3

PP3 (Not Applied)

According to standard ACMG guidelines, the rule for PP3 is: "PP3 – Multiple lines of computational evidence support a deleterious effect on the gene or gene product (e.g., conservation, splicing impact)". The evidence for this variant shows: mixed in silico predictions with overall benign tendency and no significant splicing impact. Therefore, this criterion is not applied at Not Applied strength because computational evidence does not consistently support pathogenicity.

PP4

PP4 (Not Applied)

According to standard ACMG guidelines, the rule for PP4 is: "PP4 – Patient's phenotype or family history highly specific for a disease with a single genetic etiology". The evidence for this variant shows: no phenotype or clinical presentation data. Therefore, this criterion is not applied at Not Applied strength because phenotype specificity is unknown.

PP5

PP5 (Not Applied)

According to standard ACMG guidelines, the rule for PP5 is: "PP5 – Reputable source reports variant as pathogenic, but without accessible evidence". The evidence for this variant shows: not reported in ClinVar or other expert panels. Therefore, this criterion is not applied at Not Applied strength because no reputable source classification is available.

BA1

BA1 (Not Applied)

According to standard ACMG guidelines, the rule for BA1 is: "BA1 – Allele frequency is too high for the disorder (e.g., >5%)". The evidence for this variant shows: allele frequency is 0%. Therefore, this criterion is not applied at Not Applied strength because population frequency is not above the threshold.

BS1

BS1 (Not Applied)

According to standard ACMG guidelines, the rule for BS1 is: "BS1 – Allele frequency is greater than expected for the disorder". The evidence for this variant shows: allele frequency is 0%. Therefore, this criterion is not applied at Not Applied strength because population frequency is not greater than expected.

BS2

BS2 (Not Applied)

According to standard ACMG guidelines, the rule for BS2 is: "BS2 – Observed in a healthy adult individual for a dominant (with full penetrance expected at an early age), X-linked or mitochondrial disorder with full penetrance expected at an early age". The evidence for this variant shows: no reports in healthy individuals. Therefore, this criterion is not applied at Not Applied strength because healthy adult observations are lacking.

BS3

BS3 (Not Applied)

According to standard ACMG guidelines, the rule for BS3 is: "BS3 – Well-established functional studies show no damaging effect on protein function or splicing". The evidence for this variant shows: no functional studies performed. Therefore, this criterion is not applied at Not Applied strength because functional evidence is unavailable.

BS4

BS4 (Not Applied)

According to standard ACMG guidelines, the rule for BS4 is: "BS4 – Lack of segregation in affected family members". The evidence for this variant shows: no segregation data. Therefore, this criterion is not applied at Not Applied strength because segregation analysis is not available.

BP1

BP1 (Not Applied)

According to standard ACMG guidelines, the rule for BP1 is: "BP1 – Missense variant in a gene where only loss of function causes disease". The evidence for this variant shows: POLE-associated disease mechanisms include pathogenic missense variants. Therefore, this criterion is not applied at Not Applied strength because missense is a known disease mechanism.

BP2

BP2 (Not Applied)

According to standard ACMG guidelines, the rule for BP2 is: "BP2 – Observed in trans with a pathogenic variant for a dominant disorder or in cis with a pathogenic variant". The evidence for this variant shows: no phase information. Therefore, this criterion is not applied at Not Applied strength because phase data are missing.

BP3

BP3 (Not Applied)

According to standard ACMG guidelines, the rule for BP3 is: "BP3 – In-frame deletions/insertions in a repetitive region without known function". The evidence for this variant shows: it is a missense change, not an in-frame indel. Therefore, this criterion is not applied at Not Applied strength.

BP4

BP4 (Supporting)

According to standard ACMG guidelines, the rule for BP4 is: "BP4 – Multiple lines of computational evidence suggest no impact on gene or gene product". The evidence for this variant shows: mixed in silico predictions with majority benign and SpliceAI predicts no splicing impact. Therefore, this criterion is applied at Supporting strength because computational evidence overall suggests no functional impact.

BP5

BP5 (Not Applied)

According to standard ACMG guidelines, the rule for BP5 is: "BP5 – Variant found in a case with an alternate molecular basis for disease". The evidence for this variant shows: no case reports with alternate molecular diagnoses. Therefore, this criterion is not applied at Not Applied strength.

BP6

BP6 (Not Applied)

According to standard ACMG guidelines, the rule for BP6 is: "BP6 – Reputable source reports variant as benign, but without accessible evidence". The evidence for this variant shows: not reported in any database or expert panel. Therefore, this criterion is not applied at Not Applied strength.

BP7

BP7 (Not Applied)

According to standard ACMG guidelines, the rule for BP7 is: "BP7 – Synonymous variant with no predicted impact on splicing". The evidence for this variant shows: it is a missense, not synonymous. Therefore, this criterion is not applied at Not Applied strength.